Clinical depression pathophysiology: Difference between revisions
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==Overview== | |||
==Pathophysiology== | ==Pathophysiology== | ||
==Overview== | |||
The exact pathogenesis of [disease name] is not fully understood. | |||
OR | |||
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | |||
OR | |||
[Pathogen name] is usually transmitted via the [transmission route] route to the human host. | |||
OR | |||
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | |||
OR | |||
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells]. | |||
OR | |||
The progression to [disease name] usually involves the [molecular pathway]. | |||
OR | |||
The pathophysiology of [disease/malignancy] depends on the histological subtype. | |||
==Pathophysiology== | |||
===Physiology=== | |||
The normal physiology of [name of process] can be understood as follows: | |||
===Pathogenesis=== | |||
*The exact pathogenesis of [disease name] is not completely understood. | |||
OR | |||
*It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | |||
*[Pathogen name] is usually transmitted via the [transmission route] route to the human host. | |||
*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | |||
*[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells]. | |||
*The progression to [disease name] usually involves the [molecular pathway]. | |||
*The pathophysiology of [disease/malignancy] depends on the histological subtype. | |||
==Genetics== | |||
[Disease name] is transmitted in [mode of genetic transmission] pattern. | |||
OR | |||
Genes involved in the pathogenesis of [disease name] include: | |||
*[Gene1] | |||
*[Gene2] | |||
*[Gene3] | |||
OR | |||
The development of [disease name] is the result of multiple genetic mutations such as: | |||
*[Mutation 1] | |||
*[Mutation 2] | |||
*[Mutation 3] | |||
==Associated Conditions== | |||
Conditions associated with [disease name] include: | |||
*[Condition 1] | |||
*[Condition 2] | |||
*[Condition 3] | |||
==Gross Pathology== | |||
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | |||
==Microscopic Pathology== | |||
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | |||
===Image=== | |||
[[Image:synapse.png|thumbnail|200px|center|Brain chemicals called neurotransmitters allow electrical signals to move from the axon of one nerve cell to the neuron of another. A shortage of neurotransmitters impairs brain communication.]] | [[Image:synapse.png|thumbnail|200px|center|Brain chemicals called neurotransmitters allow electrical signals to move from the axon of one nerve cell to the neuron of another. A shortage of neurotransmitters impairs brain communication.]] | ||
Revision as of 01:13, 15 June 2020
https://https://www.youtube.com/watch?v=QhukM33VLgo%7C350}} |
Clinical Depression Microchapters |
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Case Studies |
Clinical depression pathophysiology On the Web |
American Roentgen Ray Society Images of Clinical depression pathophysiology |
Risk calculators and risk factors for Clinical depression pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Please help WikiDoc by adding more content here. It's easy! Click here to learn about editing.
Overview
Pathophysiology
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Physiology
The normal physiology of [name of process] can be understood as follows:
Pathogenesis
- The exact pathogenesis of [disease name] is not completely understood.
OR
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
[Disease name] is transmitted in [mode of genetic transmission] pattern.
OR
Genes involved in the pathogenesis of [disease name] include:
- [Gene1]
- [Gene2]
- [Gene3]
OR
The development of [disease name] is the result of multiple genetic mutations such as:
- [Mutation 1]
- [Mutation 2]
- [Mutation 3]
Associated Conditions
Conditions associated with [disease name] include:
- [Condition 1]
- [Condition 2]
- [Condition 3]
Gross Pathology
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Image
References