Dyspareunia pathophysiology: Difference between revisions
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{{CMG}} {{AE}} | {{CMG}} {{AE}} | ||
==Overview== | ==Overview== | ||
[[pathophysiology]] can be considered as multifactorial, [[multisystemic]], or [[complex]]. | |||
==Pathophysiology== | ==Pathophysiology== | ||
Revision as of 01:47, 22 September 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief:
Overview
pathophysiology can be considered as multifactorial, multisystemic, or complex.
Pathophysiology
Pathophysiology of sexual pain disorders can be considered as:[1][2]
- Multifactorial
- Multisystemic
- Complex
Multifactorial:
- Biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.
Multisystemic: sexual function involves:
- Nervous system
- Endocrine system
- Vascular system
- Immunological systems
- Vaginal ecosystem: Vaginal receptiveness may be further modulated by; psychosexual, mental, interpersonal factors that may result in poor arousal with vaginal dryness. Fear of penetration, general muscular arousal secondary to anxiety, defensive contraction of the peri-vaginal muscles, leading to lifelong vaginismus.
Complex:Coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component. When It becomes chronic, the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self-protection and defense, to neuropathic, with progressive involvement of the CNS.[3]