Metabolic alkalosis classification: Difference between revisions
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Created page with "__NOTOC__ {{Metabolic alkalosis}} {{CMG}}; {{AE}} ==Overview== There is no established system for the classification of [disease name]. OR [Disease name] may be classified..." |
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{{Metabolic alkalosis}} | {{Metabolic alkalosis}} | ||
{{CMG}}; {{AE}} | {{CMG}}; {{AE}} {{MMT}} | ||
==Overview== | ==Overview== | ||
Metabolic Alkalosis can be classified according to pathophysiology, etiology and chloride responsiveness. | |||
==Classification== | ==Classification== | ||
The following classification of Metabolic Alkalosis is based on Pathophysiology: | |||
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{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis} | |||
{{Family tree | | | | |!| | | | | }} | |||
{{Family tree | | | | B01 | | | |B01= Stimulation on Collecting Duct}} | |||
{{Family tree | |,|-|-|^|-|-|.| | }} | |||
{{Family tree | C01 | | | | C02 |C01= Primary stimulation by Mineralocorticoid Excess causing HTN: •Congenital Adrenal Hyperplasia <br>•Cushing Syndrome <br>•Primary aldosteronism <br>•Renin Secreting tumors <br>•Medications(Fluoroprednisolone, Fludrocortisone) <br>•Liddle Syndrome <br>•11 beta hydroxysteroid dehydrogenase deficiency | C02= Secondary Stimulation by Chloride depletion causing normal or low blood pressure: •Vomiting <br>•NG tube intubation <br>•Congenital Chloridorrhea <br>•Ileostomy <br>•Cystic fibrosis <br>•Diuretics <br>•Bartter syndrome <br>•Gitelman syndrome <br>•Hypokalemia}} | |||
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==References== | ==References== |
Revision as of 05:03, 20 January 2021
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Marufa Marium, M.B.B.S[2]
Overview
Metabolic Alkalosis can be classified according to pathophysiology, etiology and chloride responsiveness.
Classification
The following classification of Metabolic Alkalosis is based on Pathophysiology:
{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis}Stimulation on Collecting Duct | |||||||||||||||||||
Primary stimulation by Mineralocorticoid Excess causing HTN: •Congenital Adrenal Hyperplasia •Cushing Syndrome •Primary aldosteronism •Renin Secreting tumors •Medications(Fluoroprednisolone, Fludrocortisone) •Liddle Syndrome •11 beta hydroxysteroid dehydrogenase deficiency | Secondary Stimulation by Chloride depletion causing normal or low blood pressure: •Vomiting •NG tube intubation •Congenital Chloridorrhea •Ileostomy •Cystic fibrosis •Diuretics •Bartter syndrome •Gitelman syndrome •Hypokalemia | ||||||||||||||||||