Metabolic alkalosis classification: Difference between revisions
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==Classification== | ==Classification== | ||
The following classification of Metabolic Alkalosis is based on Pathophysiology: | The following classification of Metabolic Alkalosis is based on Pathophysiology: | ||
{{Family tree/start}} | {{Family tree/start}} | ||
{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis} | {{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis} |
Revision as of 05:04, 20 January 2021
Metabolic alkalosis Microchapters |
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Metabolic alkalosis classification On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Marufa Marium, M.B.B.S[2]
Overview
Metabolic Alkalosis can be classified according to pathophysiology, etiology and chloride responsiveness.
Classification
The following classification of Metabolic Alkalosis is based on Pathophysiology:
{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis}Stimulation on Collecting Duct | |||||||||||||||||||
Primary stimulation by Mineralocorticoid Excess causing HTN: •Congenital Adrenal Hyperplasia •Cushing Syndrome •Primary aldosteronism •Renin Secreting tumors •Medications(Fluoroprednisolone, Fludrocortisone) •Liddle Syndrome •11 beta hydroxysteroid dehydrogenase deficiency | Secondary Stimulation by Chloride depletion causing normal or low blood pressure: •Vomiting •NG tube intubation •Congenital Chloridorrhea •Ileostomy •Cystic fibrosis •Diuretics •Bartter syndrome •Gitelman syndrome •Hypokalemia | ||||||||||||||||||