Metabolic alkalosis classification: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
|||
| Line 10: | Line 10: | ||
{{Family tree/start}} | {{Family tree/start}} | ||
{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis} | {{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis}} | ||
{{Family tree | | | | |!| | | | | }} | {{Family tree | | | | |!| | | | | }} | ||
{{Family tree | | | | B01 | | | |B01= Stimulation on Collecting Duct}} | {{Family tree | | | | B01 | | | |B01= Stimulation on Collecting Duct}} | ||
Revision as of 05:05, 20 January 2021
|
Metabolic alkalosis Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Metabolic alkalosis classification On the Web |
|
American Roentgen Ray Society Images of Metabolic alkalosis classification |
|
Risk calculators and risk factors for Metabolic alkalosis classification |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Marufa Marium, M.B.B.S[2]
Overview
Metabolic Alkalosis can be classified according to pathophysiology, etiology and chloride responsiveness.
Classification
The following classification of Metabolic Alkalosis is based on Pathophysiology:
| Metabolic Alkalosis | |||||||||||||||||||
| Stimulation on Collecting Duct | |||||||||||||||||||
| Primary stimulation by Mineralocorticoid Excess causing HTN: •Congenital Adrenal Hyperplasia •Cushing Syndrome •Primary aldosteronism •Renin Secreting tumors •Medications(Fluoroprednisolone, Fludrocortisone) •Liddle Syndrome •11 beta hydroxysteroid dehydrogenase deficiency | Secondary Stimulation by Chloride depletion causing normal or low blood pressure: •Vomiting •NG tube intubation •Congenital Chloridorrhea •Ileostomy •Cystic fibrosis •Diuretics •Bartter syndrome •Gitelman syndrome •Hypokalemia | ||||||||||||||||||