Sudden cardiac death urgent treatment: Difference between revisions
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=== Intervention === | |||
[[Catheter ablation]] can only be performed for patients with sustained monomorphic [[ventricular tachycardia]] based on these characteristics: | |||
*Incessant [[VT]] or electrical storm due to [[myocardial]] scar tissue | |||
* Sustained [[VT]] and recurrent [[ICD]] shock in [[ischemic heart disease]] | |||
==2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care and 2006 ACC/AHA/ESC Guidelines for Management of Patients With Cardiac Arrest (DO NOT EDIT) <ref name="pmid20956224">{{cite journal| author=Neumar RW, Otto CW, Link MS, Kronick SL, Shuster M, Callaway CW et al.| title=Part 8: adult advanced cardiovascular life support: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. | journal=Circulation | year= 2010 | volume= 122 | issue= 18 Suppl 3 | pages= S729-67 | pmid=20956224 | doi=10.1161/CIRCULATIONAHA.110.970988 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20956224 }} </ref><ref name="pmid16935995">{{cite journal| author=Zipes DP, Camm AJ, Borggrefe M, Buxton AE, Chaitman B, Fromer M et al.| title=ACC/AHA/ESC 2006 Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death: a report of the American College of Cardiology/American Heart Association Task Force and the European Society of Cardiology Committee for Practice Guidelines (writing committee to develop Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death): developed in collaboration with the European Heart Rhythm Association and the Heart Rhythm Society. | journal=Circulation | year= 2006 | volume= 114 | issue= 10 | pages= e385-484 | pmid=16935995 | doi=10.1161/CIRCULATIONAHA.106.178233 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=16935995}}</ref>== | ==2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care and 2006 ACC/AHA/ESC Guidelines for Management of Patients With Cardiac Arrest (DO NOT EDIT) <ref name="pmid20956224">{{cite journal| author=Neumar RW, Otto CW, Link MS, Kronick SL, Shuster M, Callaway CW et al.| title=Part 8: adult advanced cardiovascular life support: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. | journal=Circulation | year= 2010 | volume= 122 | issue= 18 Suppl 3 | pages= S729-67 | pmid=20956224 | doi=10.1161/CIRCULATIONAHA.110.970988 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20956224 }} </ref><ref name="pmid16935995">{{cite journal| author=Zipes DP, Camm AJ, Borggrefe M, Buxton AE, Chaitman B, Fromer M et al.| title=ACC/AHA/ESC 2006 Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death: a report of the American College of Cardiology/American Heart Association Task Force and the European Society of Cardiology Committee for Practice Guidelines (writing committee to develop Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death): developed in collaboration with the European Heart Rhythm Association and the Heart Rhythm Society. | journal=Circulation | year= 2006 | volume= 114 | issue= 10 | pages= e385-484 | pmid=16935995 | doi=10.1161/CIRCULATIONAHA.106.178233 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=16935995}}</ref>== |
Revision as of 20:56, 30 January 2021
Sudden cardiac death Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Sara Zand, M.D.[2]
Urgent Treatment
Medical Therapy
- The mainstay of therapy for patients with cardiac arrest is starting cardiopulmonary resuscitation with minimizing interruption in chest compression.[1][2]
- The rhythm should be reassessed. If the rhythm is VF orpulseless VT, the shock should be delivered immediately.
- If the rhythm is asystole or pulseless electrical activity (PEA), CPR should be resumed.
- Advanced life support (ALS) should be kept with minimizing interruption in chest compression including:
- advanced airway
- Continuous chest compressions
- after placing an advanced airway
- capnography
- IV/IO access
- vasopressors, antiarrhythmics therapy
- Correcting reversible causes including hypoxia, hypovolemia,hypothermia, hyperkalemia, hypokalemia,acidosis, tension pneumothorax, tamponade, toxins (benzodiazepines, alcohol, opiates, tricyclics, barbiturates, betablockers, calcium channel blockers)
- The followings should be considered immediately in post cardiac arrest patients:
- 12–lead ECG
- Perfusion/reperfusion in patients with acute myocardial infarction
- Oxygenation and ventilation
- Temperature control
- Treatment of reversible causes
2017AHA/ACC/HRS Guideline for management of sudden cardiac arrest and ventricular arrhythmia
Recommendations for management of cardiac arrest |
CPR (Class I, Level of Evidence A): |
❑ CPR should be done according to basic and advanced cardiovascular life support algorithms |
Amiodarone (Class I, Level of Evidence A) : |
❑ In the recurrence of ventricular arrhythmia after maximum energy shock delivery and unstable hemodynamic, amiodarone should de infused |
Direct current cardioversion : (Class I, Level of Evidence A) |
❑ In ventricular arrhythmia and unstable hemodynamic, direct current cardioversion should be delivered |
Revascularization:(Class I, Level of Evidence B) |
❑ In patients with polymorphic VT and VF and evidence of acute STEMI in ECG, coronary angiography and emergency revascularization is advised |
Wide QRS tachycardia: (Class I, Level of Evidence C) |
❑ Wide QRS tachycardia should be considered as VT if the diagnosis is unclear |
Intravenous procainamide (Class 2a, Level of Evidence A): |
❑ In hemodynamically stable VT, intravenous procainamide is recommended |
Intravenous lidocaine : (Class 2a, Level of Evidence B) |
❑ Lidocaine is recommended in witness cardiac arrest due to polymorphic VT, VF unresponsed to CPR, defibrillation or vasopressor therapy |
Intravenous betablocker : (Class 2a, Level of Evidence B) |
❑ In polymorphic VT due to myocardial ischemia, intravenous betablocker maybe helpful |
Intravenous Epinephrine : (Class 2b, Level of Evidence A) |
❑ In cardiac arrest administration of 1 mg epinephrine every 3-5 minutes during CPR is recommended |
Intravenous amiodarone : (Class 2b, Level of Evidence B) |
❑ In hemodynamic stable VT, infusion amiodarone or sotalole maybe considered |
High dose of intravenous epinephrine : (Class III , Level of Evidence A) |
❑ In cardiac arrest, administration of high dose epinephrine>1 mg bolouses is not beneficial |
Intravenous amiodarone : (Class III , Level of Evidence B) |
❑In acute myocardial infarction, prophylactic administration of lidocaine or amiodarone for prevention of VT is harmful |
Intravenous verapamil, diltiazem : (Class III , Level of Evidence C) |
❑ In a wide QRS tachycardia with unknown origin, administration of verapamil and diltiazem is harmful |
Sustained monomorphic VT | |||||||||||||||||||||||||||||||||||||||||||||||||||||||
Hemodynamic stability | |||||||||||||||||||||||||||||||||||||||||||||||||||||||
Stable | Unstable | ||||||||||||||||||||||||||||||||||||||||||||||||||||||
12-Lead ECG, history, physical exam | Dirrect current cardioversion,ACLS | ||||||||||||||||||||||||||||||||||||||||||||||||||||||
Notifying disease causing VT | Cardioversion(class1) | VT termination | |||||||||||||||||||||||||||||||||||||||||||||||||||||
Structural heart disease | Intravenous procainamide (class2a) | Yes, therapy of underlying heart disease | NO, cardioversion (class1) | ||||||||||||||||||||||||||||||||||||||||||||||||||||
NO, Ideopathic VT | Intravenous amiodarone or sotalole (class2b) | VT termination | |||||||||||||||||||||||||||||||||||||||||||||||||||||
Verapamil sensitive VT: Verapamil outflow tract VT: betablocker (class2a) | |||||||||||||||||||||||||||||||||||||||||||||||||||||||
Effective | Non effective: cardioversion | Yes,therapy of underlying heart disease | NO, Sedation ,anesthesia, reassess antiarrhythmic therapy, repeating cardioversion | ||||||||||||||||||||||||||||||||||||||||||||||||||||
Therapy to prevent recurrence of VT | No VT termination | ||||||||||||||||||||||||||||||||||||||||||||||||||||||
Catheter ablation (class1) | |||||||||||||||||||||||||||||||||||||||||||||||||||||||
Catheter ablation (class1) | Verapamil , betablocker (class2a) | ||||||||||||||||||||||||||||||||||||||||||||||||||||||
Intervention
Catheter ablation can only be performed for patients with sustained monomorphic ventricular tachycardia based on these characteristics:
- Incessant VT or electrical storm due to myocardial scar tissue
- Sustained VT and recurrent ICD shock in ischemic heart disease
2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care and 2006 ACC/AHA/ESC Guidelines for Management of Patients With Cardiac Arrest (DO NOT EDIT) [3][4]
Class III |
"1. Adenosine should not be given for unstable or for irregular or polymorphic ventricular tachycardias, as it may cause degeneration of the arrhythmia to VF. (Level of Evidence: C)" |
"1. Verapamil is contraindicated for wide complex tachycardias unless known to be of supraventricular origin. (Level of Evidence: B)" |
"1. If one of these antiarrhythmic agents is given, a second agent should not be given without expert consultation. (Level of Evidence: B)" |
Class IIa |
" 1. Cardioversion with monophasic waveforms should begin at 200 J and increase in stepwise fashion if not successful. (Level of Evidence: B) " |
" 2. If the etiology of the rhythm cannot be determined, the rate is regular, and the QRS is monomorphic, recent evidence suggests that IV adenosine is relatively safe for both treatment and diagnosis.[5] (Level of Evidence: B) " |
" 3. If IV antiarrhythmics are administered, procainamide can be considered. (Level of Evidence: B) " |
" 4. If antiarrhythmic therapy is unsuccessful, cardioversion or expert consultation should be considered. (Level of Evidence: C) " |
Class IIb |
" 1. Monomorphic VT with a pulse responds well to monophasic or biphasic waveform cardioversion (synchronized) shocks at initial energies of 100 J. If there is no response to the first shock, it may be reasonable to increase the dose in a stepwise fashion. (Level of Evidence: C) " |
" 2. Precordial thump may be considered for patients with witnessed, monitored, unstable ventricular tachycardia if a defibrillator is not immediately ready for use. (Level of Evidence: C) " |
" 3. If IV antiarrhythmics are administered, amiodarone or sotalol can be considered. (Level of Evidence: B) " |
Management of Cardiac Arrest (DO NOT EDIT) [4]
Class I |
"1 After establishing the presence of definite, suspected, or impending cardiac arrest, the first priority should be activation of a response team capable of identifying the specific mechanism and carrying out prompt intervention. (Level of Evidence: B) " |
"2 Cardiopulmonary resuscitation (CPR) should be implemented immediately after contacting a response team. (Level of Evidence: A) " |
"3 In an out-of-hospital setting, if an AED is available, it should be applied immediately and shock therapy administered according to the algorithms contained in the documents on CPR (334,335) developed by the AHA in association with the International Liaison Committee on Resuscitation (ILCOR) and/or the European Resuscitation Council (ERC).[6][7] (Level of Evidence: C) " |
"4 For victims with ventricular tachyarrhythmic mechanisms of cardiac arrest, when recurrences occur after a maximally defibrillating shock (generally 360 J for monophasic defibrillators), intravenous amiodarone should be the preferred antiarrhythmic drug for attempting a stable rhythm after further defibrillations. (Level of Evidence: B) " |
"5 For recurrent ventricular tachyarrhythmias or nontachyarrhythmic mechanisms of cardiac arrest, it is recommended to follow the algorithms contained in the documents on CPR (334,335) developed by the AHA in association with ILCOR and/or the ERC.[6][7] (Level of Evidence: C) " |
"6 Reversible causes and factors contributing to cardiac arrest should be managed during advanced life support, including management of hypoxia, electrolyte disturbances, mechanical factors, and volume depletion. (Level of Evidence: C) " |
Class IIa |
"1 For response times greater than or equal to 5 min, a brief (less than 90 to 180 s) period of CPR is reasonable prior to attempting defibrillation. (Level of Evidence: B)" |
Class IIb |
"1 A single precordial thump may be considered by health care professional providers when responding to a witnessed cardiac arrest. (Level of Evidence: C)" |
Management of Cardiac Arrest in Athletes (DO NOT EDIT) [4]
Class I |
"1 Preparticipation history and physical examination, including family history of premature or SCD and specific evidence of cardiovascular diseases such as cardiomyopathies and ion channel abnormalities, is recommended in athletes. (Level of Evidence: C)" |
"2. Athletes presenting with rhythm disorders, structural heart disease, or other signs or symptoms suspicious for cardiovascular disorders should be evaluated as any other patient but with recognition of the potential uniqueness of their activity. (Level of Evidence: C)" |
"3. Athletes presenting with syncope should be carefully evaluated to uncover underlying cardiovascular disease or rhythm disorder. (Level of Evidence: B)" |
"4. Athletes with serious symptoms should cease competition while cardiovascular abnormalities are being fully evaluated. (Level of Evidence: C)" |
Class IIb |
"1. Twelve-lead ECG and possibly echocardiography may be considered as preparticipation screening for heart disorders in athletes. (Level of Evidence: B)" |
References
- ↑ Priori, Silvia G.; Blomström-Lundqvist, Carina; Mazzanti, Andrea; Blom, Nico; Borggrefe, Martin; Camm, John; Elliott, Perry Mark; Fitzsimons, Donna; Hatala, Robert; Hindricks, Gerhard; Kirchhof, Paulus; Kjeldsen, Keld; Kuck, Karl-Heinz; Hernandez-Madrid, Antonio; Nikolaou, Nikolaos; Norekvål, Tone M.; Spaulding, Christian; Van Veldhuisen, Dirk J. (2015). "2015 ESC Guidelines for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death". European Heart Journal. 36 (41): 2793–2867. doi:10.1093/eurheartj/ehv316. ISSN 0195-668X.
- ↑ Al-Khatib, Sana M.; Stevenson, William G.; Ackerman, Michael J.; Bryant, William J.; Callans, David J.; Curtis, Anne B.; Deal, Barbara J.; Dickfeld, Timm; Field, Michael E.; Fonarow, Gregg C.; Gillis, Anne M.; Granger, Christopher B.; Hammill, Stephen C.; Hlatky, Mark A.; Joglar, José A.; Kay, G. Neal; Matlock, Daniel D.; Myerburg, Robert J.; Page, Richard L. (2018). "2017 AHA/ACC/HRS Guideline for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death". Circulation. 138 (13). doi:10.1161/CIR.0000000000000549. ISSN 0009-7322.
- ↑ Neumar RW, Otto CW, Link MS, Kronick SL, Shuster M, Callaway CW; et al. (2010). "Part 8: adult advanced cardiovascular life support: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care". Circulation. 122 (18 Suppl 3): S729–67. doi:10.1161/CIRCULATIONAHA.110.970988. PMID 20956224.
- ↑ 4.0 4.1 4.2 Zipes DP, Camm AJ, Borggrefe M, Buxton AE, Chaitman B, Fromer M; et al. (2006). "ACC/AHA/ESC 2006 Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death: a report of the American College of Cardiology/American Heart Association Task Force and the European Society of Cardiology Committee for Practice Guidelines (writing committee to develop Guidelines for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death): developed in collaboration with the European Heart Rhythm Association and the Heart Rhythm Society". Circulation. 114 (10): e385–484. doi:10.1161/CIRCULATIONAHA.106.178233. PMID 16935995.
- ↑ Staudinger T, Brugger S, Röggla M, Rintelen C, Atherton GL, Johnson JC; et al. (1994). "[Comparison of the Combitube with the endotracheal tube in cardiopulmonary resuscitation in the prehospital phase]". Wien Klin Wochenschr. 106 (13): 412–5. PMID 8091765.
- ↑ 6.0 6.1 "2005 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care". Circulation. 112 (24 Suppl): IV1–203. 2005. doi:10.1161/CIRCULATIONAHA.105.166550. PMID 16314375. Retrieved 2012-11-05. Unknown parameter
|month=
ignored (help) - ↑ 7.0 7.1 Nolan JP, Deakin CD, Soar J, Böttiger BW, Smith G (2005). "European Resuscitation Council guidelines for resuscitation 2005. Section 4. Adult advanced life support". Resuscitation. 67 Suppl 1: S39–86. doi:10.1016/j.resuscitation.2005.10.009. PMID 16321716. Retrieved 2012-11-05. Unknown parameter
|month=
ignored (help)