Anti-NMDA receptor encephalitis MRI: Difference between revisions
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==Key MRI Findings for Anti-NMDA Receptor Encephalitis== | ==Key MRI Findings for Anti-NMDA Receptor Encephalitis== | ||
In fifty percent of instances, magnetic resonance (MR) imaging of the brain is unremarkable. In the remaining 50%, MR Imaging may demonstrate nonspecific T2 or fluid-attenuated inversion recovery (FLAIR) signal hyperintensity in the hippocampus; cerebellar, frontobasal, or insular cortex; basal ganglia; brainstem; and, rarely, the spinal cord. | |||
Anti-NMDAR encephalitis is notoriously difficult to diagnose based only on neuroimaging characteristics, as there are no discernible patterns of brain involvement | |||
==Examples of MRI Findings for Anti-NMDA Receptor Encephalitis== | ==Examples of MRI Findings for Anti-NMDA Receptor Encephalitis== |
Revision as of 11:07, 11 February 2023
Anti-NMDA receptor encephalitis Microchapters |
Differentiating Anti-NMDA receptor encephalitis from Other Diseases |
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Diagnosis |
Treatment |
Case Studies |
Anti-NMDA receptor encephalitis MRI On the Web |
American Roentgen Ray Society Images of Anti-NMDA receptor encephalitis MRI |
Directions to Hospitals Treating Anti-NMDA receptor encephalitis |
Risk calculators and risk factors for Anti-NMDA receptor encephalitis MRI |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Key MRI Findings for Anti-NMDA Receptor Encephalitis
In fifty percent of instances, magnetic resonance (MR) imaging of the brain is unremarkable. In the remaining 50%, MR Imaging may demonstrate nonspecific T2 or fluid-attenuated inversion recovery (FLAIR) signal hyperintensity in the hippocampus; cerebellar, frontobasal, or insular cortex; basal ganglia; brainstem; and, rarely, the spinal cord.
Anti-NMDAR encephalitis is notoriously difficult to diagnose based only on neuroimaging characteristics, as there are no discernible patterns of brain involvement