Anti-NMDA receptor encephalitis MRI: Difference between revisions
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In the remaining 50%, MR Imaging may demonstrate nonspecific T2 or fluid-attenuated inversion recovery (FLAIR) signal hyperintensity in the hippocampus; cerebellar, frontobasal, or insular cortex; basal ganglia; brainstem; and, rarely, the spinal cord. | In the remaining 50%, MR Imaging may demonstrate nonspecific T2 or fluid-attenuated inversion recovery (FLAIR) signal hyperintensity in the hippocampus; cerebellar, frontobasal, or insular cortex; basal ganglia; brainstem; and, rarely, the spinal cord. | ||
The presence of bilateral medial temporal lobe hyperintensities, in particular, has been reported in some cases. | |||
Anti-NMDAR encephalitis is notoriously difficult to diagnose based only on neuroimaging characteristics, as there are no discernible patterns of brain involvement | Anti-NMDAR encephalitis is notoriously difficult to diagnose based only on neuroimaging characteristics, as there are no discernible patterns of brain involvement | ||
Revision as of 04:50, 13 May 2023
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Anti-NMDA receptor encephalitis Microchapters |
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Differentiating Anti-NMDA receptor encephalitis from Other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Anti-NMDA receptor encephalitis MRI On the Web |
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American Roentgen Ray Society Images of Anti-NMDA receptor encephalitis MRI |
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Directions to Hospitals Treating Anti-NMDA receptor encephalitis |
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Risk calculators and risk factors for Anti-NMDA receptor encephalitis MRI |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Key MRI Findings for Anti-NMDA Receptor Encephalitis
In fifty percent of instances, magnetic resonance (MR) imaging of the brain is unremarkable. MRI scans may show non-specific abnormalities, with temporal lobe involvement being one of the most commonly reported findings. These findings are not specific to anti-NMDA encephalitis and can also be seen in other conditions.
T2-Weighted and Fluid-Attenuated Inversion Recovery (FLAIR) Sequences: In the remaining 50%, MR Imaging may demonstrate nonspecific T2 or fluid-attenuated inversion recovery (FLAIR) signal hyperintensity in the hippocampus; cerebellar, frontobasal, or insular cortex; basal ganglia; brainstem; and, rarely, the spinal cord.
The presence of bilateral medial temporal lobe hyperintensities, in particular, has been reported in some cases.
Anti-NMDAR encephalitis is notoriously difficult to diagnose based only on neuroimaging characteristics, as there are no discernible patterns of brain involvement