Anti-NMDA receptor encephalitis overview: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
Anti-NMDA receptor encephalitis is an autoimmune disorder characterized by the presence of autoantibodies directed against the GluN1 subunit of the NMDA receptor. The presence of tumors, particularly ovarian teratomas in women and testicular teratomas or lung carcinomas in men, is frequently associated with the condition. Dysregulation of NMDA receptors contributes to the development of diverse neurological and psychiatric symptoms. | |||
==Causes== | ==Causes== | ||
Revision as of 17:46, 13 May 2023
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Anti-NMDA receptor encephalitis Microchapters |
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Differentiating Anti-NMDA receptor encephalitis from Other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Anti-NMDA receptor encephalitis overview On the Web |
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Risk calculators and risk factors for Anti-NMDA receptor encephalitis overview |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Historical Perspective
Anti-NMDA encephalitis, initially documented in a case report dating back to 1830, is an autoimmune condition marked by neuropsychiatric manifestations. In some instances, the presence of an ovarian teratoma is observed. The case of Susannah Cahalan played a significant role in raising public awareness regarding this disorder.
Classification
Anti-NMDA encephalitis can be categorized into three distinct subtypes based on the timing of onset and duration of the illness.
Pathophysiology
Anti-NMDA receptor encephalitis is an autoimmune disorder characterized by the presence of autoantibodies directed against the GluN1 subunit of the NMDA receptor. The presence of tumors, particularly ovarian teratomas in women and testicular teratomas or lung carcinomas in men, is frequently associated with the condition. Dysregulation of NMDA receptors contributes to the development of diverse neurological and psychiatric symptoms.