Hyperaldosteronism: Difference between revisions
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'''Hyperaldosteronism''', also '''aldosteronism''', is a [[medical condition]] where too much [[aldosterone]] is produced by the [[adrenal glands]], which can lead to lowered levels of [[potassium]] in [[blood]]. | '''Hyperaldosteronism''', also '''aldosteronism''', is a [[medical condition]] where too much [[aldosterone]] is produced by the [[adrenal glands]], which can lead to lowered levels of [[potassium]] in [[blood]]. | ||
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==Types== | ==Types== | ||
* {{ICD10|E|26|0|e|20}}: Primary hyperaldosteronism, often caused by an adrenal cortical adenoma (a tumor), is also known as [[Conn's syndrome]]. | * {{ICD10|E|26|0|e|20}}: Primary hyperaldosteronism, often caused by an adrenal cortical adenoma (a tumor), is also known as [[Conn's syndrome]]. | ||
* {{ICD10|E|26|1|e|20}}: Secondary hyperaldosteronism is due to overactivity of the [[renin-angiotensin system]]. | * {{ICD10|E|26|1|e|20}}: Secondary hyperaldosteronism is due to overactivity of the [[renin-angiotensin system]]. | ||
In [[endocrinology]], the terms ''primary'' and ''secondary'' are used to describe the abnormality (e.g. elevated [[aldosterone]]) in relation to the defect, i.e. the tumor's location. The adjective ''primary'' refers to an abnormality that directly leads to pathology, i.e. aldosteronism is caused by the apparatus that generates [[aldosterone]]. ''Secondary'' refers to an abnormality that indirectly results in pathology through a predictable physiologic pathway, i.e. a [[renin]] producing tumor leads to increased [[aldosterone]], as the body's [[aldosterone]] production is normally regulated by [[renin]] levels. | In [[endocrinology]], the terms ''primary'' and ''secondary'' are used to describe the abnormality (e.g. elevated [[aldosterone]]) in relation to the defect, i.e. the tumor's location. The adjective ''primary'' refers to an abnormality that directly leads to pathology, i.e. aldosteronism is caused by the apparatus that generates [[aldosterone]]. ''Secondary'' refers to an abnormality that indirectly results in pathology through a predictable physiologic pathway, i.e. a [[renin]] producing tumor leads to increased [[aldosterone]], as the body's [[aldosterone]] production is normally regulated by [[renin]] levels. | ||
==Symptoms== | ==Symptoms== | ||
It can be [[asymptomatic]], but the following symptoms can be present | It can be [[asymptomatic]], but the following symptoms can be present | ||
* [[Fatigue (physical)|Fatigue]] | * [[Fatigue (physical)|Fatigue]] | ||
* [[Headache]] | * [[Headache]] | ||
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[[Category:Diseases]] | [[Category:Diseases]] | ||
[[Category:Endocrinology]] | [[Category:Endocrinology]] | ||
[[Category:Mature chapter]] | |||
Revision as of 02:00, 29 July 2011
| Hyperaldosteronism | |
 | |
|---|---|
| Aldosterone | |
| ICD-10 | E26 |
| ICD-9 | 255.1 |
| DiseasesDB | 6187 |
| MedlinePlus | 000330 |
| eMedicine | radio/354 |
| MeSH | D006929 |
For patient information click here
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Hyperaldosteronism, also aldosteronism, is a medical condition where too much aldosterone is produced by the adrenal glands, which can lead to lowered levels of potassium in blood.
Types
- E26.0: Primary hyperaldosteronism, often caused by an adrenal cortical adenoma (a tumor), is also known as Conn's syndrome.
- E26.1: Secondary hyperaldosteronism is due to overactivity of the renin-angiotensin system.
In endocrinology, the terms primary and secondary are used to describe the abnormality (e.g. elevated aldosterone) in relation to the defect, i.e. the tumor's location. The adjective primary refers to an abnormality that directly leads to pathology, i.e. aldosteronism is caused by the apparatus that generates aldosterone. Secondary refers to an abnormality that indirectly results in pathology through a predictable physiologic pathway, i.e. a renin producing tumor leads to increased aldosterone, as the body's aldosterone production is normally regulated by renin levels.
Symptoms
It can be asymptomatic, but the following symptoms can be present
- Fatigue
- Headache
- High blood pressure
- Hypokalemia
- Intermittent or temporary paralysis
- Muscle spasms
- Muscle weakness
- Numbness
- Polyuria
- Polydipsia
- Tingling
Complete Differential Diagnosis of the Causes of Hyperaldosteronism
(By organ system)
|
Cardiovascular |
No underlying causes |
| Chemical / poisoning | No underlying causes |
| Dermatologic | No underlying causes |
| Drug Side Effect | No underlying causes |
| Ear Nose Throat | No underlying causes |
| Endocrine | No underlying causes |
| Environmental | No underlying causes |
| Gastroenterologic | No underlying causes |
| Genetic | No underlying causes |
| Hematologic | No underlying causes |
| Iatrogenic | No underlying causes |
| Infectious Disease | No underlying causes |
| Musculoskeletal / Ortho | No underlying causes |
| Neurologic | No underlying causes |
| Nutritional / Metabolic | No underlying causes |
| Obstetric/Gynecologic | No underlying causes |
| Oncologic | No underlying causes |
| Opthalmologic | No underlying causes |
| Overdose / Toxicity | No underlying causes |
| Psychiatric | No underlying causes |
| Pulmonary | No underlying causes |
| Renal / Electrolyte | No underlying causes |
| Rheum / Immune / Allergy | No underlying causes |
| Sexual | No underlying causes |
| Trauma | No underlying causes |
| Urologic | No underlying causes |
| Miscellaneous | No underlying causes |
See also
External links
de:Hyperaldosteronismus
it:Iperaldosteronismo