Pregnancy and heart disease congenital heart disease: Difference between revisions
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{{Pregnancy and heart disease}} | {{Pregnancy and heart disease}} | ||
{{CMG}}; '''Associate Editor-In-Chief:''' {{AC}} | {{CMG}}; '''Associate Editor-In-Chief:''' {{AC}} [[Priyamvada Singh|Priyamvada Singh, M.B.B.S.]] [mailto:psingh@perfuse.org]; {{CZ}} '''Assistant Editor(s)-In-Chief:''' [[Kristin Feeney|Kristin Feeney, B.S.]] [mailto:kfeeney@perfuse.org] | ||
==Overview== | ==Overview== | ||
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==[[Atrial septal defect and pregnancy|Atrial Septal Defect]]== | ==[[Atrial septal defect and pregnancy|Atrial Septal Defect]]== | ||
Pregnancy causes an increase in [[cardiac output]] and [[stroke volume]]. This can cause an increased [[left-to-right shunt]]ing of blood. Despite the increased workload on [[heart]], females with isolated asymptomatic [[atrial septal defect]]s tolerate the pregnancy well. Pregnant females with an atrial septal defect may have increased frequencies of some complications for instance [[arrhythmias]], [[thromboembolism]], and [[bleeding]]. Despite this, there are no studies suggesting that pregnancy requires different indications for closure in pregnant females with atrial septal defect compared to a non-pregnant female with [[atrial septal defect]]. The ACC/[[AHA]] guidelines, however, do dictate clear deviations in course of treatment in certain special circumstances. According to ACC/AHA guidelines pregnancy could be harmful in females with [[atrial septal defect]] and severe [[pulmonary hypertension]] ([[Eisenmenger syndrome]]) | |||
==References== | ==References== | ||
Revision as of 01:23, 23 September 2011
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Cardiac disease in pregnancy Microchapters |
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Diagnosis |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Anjan K. Chakrabarti, M.D. [2] Priyamvada Singh, M.B.B.S. [3]; Cafer Zorkun, M.D., Ph.D. [4] Assistant Editor(s)-In-Chief: Kristin Feeney, B.S. [5]
Overview
The most common maternal cardiac disease in Western societies is now congenital heart disease. This includes a wide spectrum of abnormalities, which are delineated in this section. This is largely due in part to the fact that children born with congenital heart defects in Western societies are living to the ages at which they can bear children. This section will not detail those patients that have repaired congenital defects, which are highlighted in ****.
Atrial Septal Defect
Pregnancy causes an increase in cardiac output and stroke volume. This can cause an increased left-to-right shunting of blood. Despite the increased workload on heart, females with isolated asymptomatic atrial septal defects tolerate the pregnancy well. Pregnant females with an atrial septal defect may have increased frequencies of some complications for instance arrhythmias, thromboembolism, and bleeding. Despite this, there are no studies suggesting that pregnancy requires different indications for closure in pregnant females with atrial septal defect compared to a non-pregnant female with atrial septal defect. The ACC/AHA guidelines, however, do dictate clear deviations in course of treatment in certain special circumstances. According to ACC/AHA guidelines pregnancy could be harmful in females with atrial septal defect and severe pulmonary hypertension (Eisenmenger syndrome)