Mixed angina pectoris: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
* Dynamic [[vasoconstriction]] which is superimposed on fixed atherosclerotic coronary artery obstruction has been postulated as the underlying pathophysiologic mechanism for the | * Dynamic [[vasoconstriction]] which is superimposed on fixed atherosclerotic coronary artery obstruction has been postulated as the underlying pathophysiologic mechanism for the changes in exercise threshold in variable angina pectoris. | ||
==Diagnosis== | ==Diagnosis== | ||
Revision as of 19:41, 14 May 2012
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Chronic stable angina Microchapters | ||
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Classification | ||
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Differentiating Chronic Stable Angina from Acute Coronary Syndromes | ||
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Diagnosis | ||
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Alternative Therapies for Refractory Angina | ||
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Discharge Care | ||
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Guidelines for Asymptomatic Patients | ||
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Case Studies | ||
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Mixed angina pectoris On the Web | ||
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]
Overview
Mixed or variable threshold angina pectoris is a syndrome in which there is substantial variation in the magnitude of physical activity that induces anginal chest pain.
Pathophysiology
- Dynamic vasoconstriction which is superimposed on fixed atherosclerotic coronary artery obstruction has been postulated as the underlying pathophysiologic mechanism for the changes in exercise threshold in variable angina pectoris.
Diagnosis
Symptoms
- The essential clinical feature of mixed angina is a substantial variation in the degree of physical activity that induces angina.
- These group of patients may also experience rest or nocturnal angina on certain occasions.
- Angina may also occur on exposure to cold, during emotional stress, or after meals.
Treatment
- The treatment is the same as chronic stable angina.