Neurocardiogenic syncope tilt table testing: Difference between revisions
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==Overview== | ==Overview== | ||
The head-up tilt table test mimics the orthostatic stress. The orthostatic stress results in maximal venous pooling, central hypovolaemia and peripheral provocation of vasovagal syncope. | The head-up tilt table test mimics the orthostatic stress. The orthostatic stress results in maximal venous pooling, central hypovolaemia and peripheral provocation of vasovagal syncope. | ||
==Role in diagnosis of Neurocardiogenic Shock== | |||
In-spite of the test being positive in patients with neurocardiogenic shock, it is not considered as a gold standard for diagnosis because of its limited sensitivity, specificity and reproducibility. | |||
Depending on patient selection and protocol, the test has high sensitivity (upto 80%) but low specificity. | |||
Revision as of 02:06, 21 May 2012
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Neurocardiogenic Syncope Microchapters |
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Differentiating Neurocardiogenic Syncope From Other Diseases |
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Diagnosis |
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Treatment |
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Neurocardiogenic syncope tilt table testing On the Web |
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American Roentgen Ray Society Images of Neurocardiogenic syncope tilt table testing |
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Risk calculators and risk factors for Neurocardiogenic syncope tilt table testing |
Overview
The head-up tilt table test mimics the orthostatic stress. The orthostatic stress results in maximal venous pooling, central hypovolaemia and peripheral provocation of vasovagal syncope.
Role in diagnosis of Neurocardiogenic Shock
In-spite of the test being positive in patients with neurocardiogenic shock, it is not considered as a gold standard for diagnosis because of its limited sensitivity, specificity and reproducibility.
Depending on patient selection and protocol, the test has high sensitivity (upto 80%) but low specificity.