Pseudo-Bartter syndrome: Difference between revisions
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==Diagnosis== | ==Diagnosis== | ||
===Laboratory Findings=== | ===Laboratory Findings=== | ||
The disorder is characterized by [[ | The disorder is characterized by | ||
*[[Hypokalemic-hypochloremic alkalosis]] | |||
*Hyperactivity of [[renin-angiotensin-aldosterone system]] | |||
*Increased [[aldosterone]] | |||
*Normal [[blood pressure]] ([[normotension]]) | |||
*Inactivity of [[angiotensin II]] | |||
*Increased urinary [[PGE2]] | |||
*Increased [[ANP]] | |||
==References== | |||
{{Reflist|2}} | |||
{{Nephrology | {{Nephrology}} | ||
[[Category:Nephrology]] | [[Category:Nephrology]] |
Revision as of 17:21, 14 July 2012
Pseudo-Bartter syndrome |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
A condition that mimics Bartter's syndrome which is due instead to furosemide treatment.
Diagnosis
Laboratory Findings
The disorder is characterized by
- Hypokalemic-hypochloremic alkalosis
- Hyperactivity of renin-angiotensin-aldosterone system
- Increased aldosterone
- Normal blood pressure (normotension)
- Inactivity of angiotensin II
- Increased urinary PGE2
- Increased ANP