Beer potomania: Difference between revisions
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==[[Beer potomania risk factors|Risk Factors]]== | ==[[Beer potomania risk factors|Risk Factors]]== | ||
==[[Beer potomania natural history, complications | ==[[Beer potomania natural history, complications and prognosis|Natural History, Complications, and Prognosis]]== | ||
=== Differential Diagnosis === | === Differential Diagnosis === | ||
See the differential of [[hyponatremia]] | See the differential of [[hyponatremia]] |
Revision as of 14:44, 22 August 2012
Beer potomania Microchapters |
Diagnosis |
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Treatment |
Case Studies |
Beer potomania On the Web |
American Roentgen Ray Society Images of Beer potomania |
Editor-in-Chief: Suresh Samson, M.D., Yale Bridgeport Hospital
Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Beer potomania from other Diseases
Epidemiology and Demographics
Risk Factors
Natural History, Complications, and Prognosis
Differential Diagnosis
See the differential of hyponatremia
Electrolyte and Biomarker Studies
A value of Uosm < 100 mOsm/kg is indicative of a complete and appropriate suppression of antidiuretic hormone (ADH) secretion, a finding seen with either primary polydipsia (including ‘beer potomania’) or reset osmostat. Criteria used to diagnose the ‘beer potomania’ syndrome include
- A history of binge beer drinking
- Poor dietary intake
- Decreased serum sodium levels
- Low serum and urine osmolality
- Absence of other known causes of hyponatremia