Meningococcemia pathophysiology: Difference between revisions
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Revision as of 14:46, 27 August 2012
Meningococcemia Microchapters |
Diagnosis |
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Treatment |
Case Studies |
Meningococcemia pathophysiology On the Web |
American Roentgen Ray Society Images of Meningococcemia pathophysiology |
Risk calculators and risk factors for Meningococcemia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]
Overview
Pathophysiology
- Transmission through respiratory secretions from a nasopharyngeal carrier case-patient.
- Shock is due to lipooligosaccharide which is a potent toxin. This toxin initiates release of inflammatory cytokines, reactive oxygen radicals, prostaglandins, arachidonic acid, complement activated products, platelet aggregating factor, and perhaps nitric oxide.