Accelerated idioventricular rhythm definition: Difference between revisions
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==Overview== | ==Overview== | ||
Accelerated Idioventricular Rhythms are ectopic ventricular rhythms at rates between 40 bpm and 100 to 120 bpm. The ventricular origin of this rhythm can be demonstrated by the usual EKG criteria which include [[AV dissociation]], [[fusion beats]], and capture complexes. | Accelerated Idioventricular Rhythms are ectopic ventricular rhythms at rates between 40 bpm and 100 to 120 bpm. The ventricular origin of this rhythm can be demonstrated by the usual EKG criteria which include [[AV dissociation]], [[fusion beats]], and capture complexes. | ||
The rate of cardiac contraction is determined by the intrinsic rate of depolarisation of the cardiac cells. In normal hearts the [[sinoatrial node]] in the atria depolarises at a rate of 70 beats per minute. This suppresses the intrinsic depolarisation of the other parts of the heart. | The rate of cardiac contraction is determined by the intrinsic rate of depolarisation of the cardiac cells. In normal hearts the [[sinoatrial node]] in the atria depolarises at a rate of 70 beats per minute. This suppresses the intrinsic depolarisation of the other parts of the heart. | ||
Revision as of 22:49, 3 September 2012
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Accelerated idioventricular rhythm Microchapters |
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Differentiating Accelerated idioventricular rhythm from other Diseases |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Accelerated Idioventricular Rhythms are ectopic ventricular rhythms at rates between 40 bpm and 100 to 120 bpm. The ventricular origin of this rhythm can be demonstrated by the usual EKG criteria which include AV dissociation, fusion beats, and capture complexes.
The rate of cardiac contraction is determined by the intrinsic rate of depolarisation of the cardiac cells. In normal hearts the sinoatrial node in the atria depolarises at a rate of 70 beats per minute. This suppresses the intrinsic depolarisation of the other parts of the heart.
The accelerated idioventricular rhythm occurs when depolarisation rate of a normally suppressed focus increases to above that of the "higher order" focuses (the sinoatrial node and the atrioventricular node). This most commonly occurs in the setting of a sinus bradycardia.