Gastric antral vascular ectasia pathophysiology: Difference between revisions
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Created page with "__NOTOC__ {{Gastric antral vascular ectasia}} {{CMG}} ==Overview== ==Pathophysiology== GAVE is characterized by dilated capillaries in the lamina propria with fibrin thromb..." |
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==Pathophysiology== | ==Pathophysiology== | ||
GAVE is characterized by dilated capillaries in the lamina propria with fibrin thrombi. | GAVE is characterized by dilated capillaries in the lamina propria with fibrin thrombi. | ||
===Gross Pathology=== | |||
[[Image:Gastric_antral_vascular_ectasia_gross_image.JPG|thumb|left|Endoscopic image of gastric antral vascular ectasia]] | |||
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===Microscopic Pathology=== | ===Microscopic Pathology=== | ||
Latest revision as of 16:28, 5 September 2012
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Gastric antral vascular ectasia Microchapters |
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Differentiating Gastric antral vascular ectasia from other Diseases |
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Gastric antral vascular ectasia pathophysiology On the Web |
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Risk calculators and risk factors for Gastric antral vascular ectasia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
GAVE is characterized by dilated capillaries in the lamina propria with fibrin thrombi.
Gross Pathology
Microscopic Pathology
Asociated Conditions
GAVE is associated with a number of conditions, including
- Portal hypertension
- Chronic renal failure
- Collagen vascular diseases, particularly scleroderma
- Pernicious anemia
- Liver cirrhosis
- Chronic renal failure
- Bone marrow transplantation