Hypogonadism pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
Gonads have two functions: to produce [[hormone]]s ([[testosterone]], [[estradiol]], [[antimullerian hormone]], [[progesterone]], [[inhibin|inhibin B]]), [[activin]] and to produce [[gamete]]s ([[Ovum|egg]]s or [[Spermatozoon|sperm]]). Deficiency of sex hormones can result in defective primary or secondary sexual development, or withdrawal effects (e.g., premature [[menopause]]) in adults. Defective egg or sperm development results in [[infertility]]. The term hypogonadism is usually applied to permanent rather than transient or reversible defects, and usually implies deficiency of reproductive hormones, with or without [[fertility]] defects. The term is less commonly used for infertility without hormone deficiency. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 23:33, 18 September 2012
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Hypogonadism Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Hypogonadism pathophysiology On the Web |
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American Roentgen Ray Society Images of Hypogonadism pathophysiology |
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Risk calculators and risk factors for Hypogonadism pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Gonads have two functions: to produce hormones (testosterone, estradiol, antimullerian hormone, progesterone, inhibin B), activin and to produce gametes (eggs or sperm). Deficiency of sex hormones can result in defective primary or secondary sexual development, or withdrawal effects (e.g., premature menopause) in adults. Defective egg or sperm development results in infertility. The term hypogonadism is usually applied to permanent rather than transient or reversible defects, and usually implies deficiency of reproductive hormones, with or without fertility defects. The term is less commonly used for infertility without hormone deficiency.