Thrombophilia pathophysiology: Difference between revisions
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(Created page with "__NOTOC__ {{Thrombophilia}} {{CMG}} ==Overview== ==References== {{reflist|2}} {{WH}} {{WS}} Category:Disease Category:Hematology") |
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
The Virchow's triad has been described classically as the patho-physiologic mechanism responsible for any thrombosis, which includes 3 components: | |||
* [[Endothelial dysfunction]] | |||
* [[Venous stasis]] | |||
* [[Hypercoaguability]] | |||
The mechanism of thrombophilia involves affecting the pathway of thrombosis<ref name="pmid11309638">{{cite journal |author=Seligsohn U, Lubetsky A |title=Genetic susceptibility to venous thrombosis |journal=N. Engl. J. Med. |volume=344 |issue=16 |pages=1222–31 |year=2001 |month=April |pmid=11309638 |doi=10.1056/NEJM200104193441607 |url=}}</ref>: | |||
[[Image: Figure_thrombophilia_mechanism.jpg]] | |||
Adapted from: N Engl J Med. 2001 Apr 19;344(16):1222-31. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 13:21, 21 September 2012
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Thrombophilia Microchapters |
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Thrombophilia pathophysiology On the Web |
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Risk calculators and risk factors for Thrombophilia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
The Virchow's triad has been described classically as the patho-physiologic mechanism responsible for any thrombosis, which includes 3 components:
The mechanism of thrombophilia involves affecting the pathway of thrombosis[1]:
Adapted from: N Engl J Med. 2001 Apr 19;344(16):1222-31.