Bronchospasm pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
Bronchospasm is caused by the release ([[degranulation]]) of substances from [[mast cells]] or [[basophil]]s under the influence of [[anaphylatoxin]]s. The overactivity of the bronchioles' muscle is a result of exposure to a stimulus which under normal circumstances would cause little or no response. The resulting constriction and inflammation causes a narrowing of the airways and an increase in [[mucus]] production; this reduces the amount of [[oxygen]] that is available to the individual causing breathlessness, coughing and [[hypoxia (medical)|hypoxia]]. | Bronchospasm is caused by the release ([[degranulation]]) of substances from [[mast cells]] or [[basophil]]s under the influence of [[anaphylatoxin]]s. The overactivity of the bronchioles' muscle is a result of exposure to a stimulus which under normal circumstances would cause little or no response. The resulting constriction and inflammation causes a narrowing of the airways and an increase in [[mucus]] production; this reduces the amount of [[oxygen]] that is available to the individual causing breathlessness, coughing and [[hypoxia (medical)|hypoxia]]. | ||
Revision as of 18:06, 25 September 2012
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Bronchospasm is caused by the release (degranulation) of substances from mast cells or basophils under the influence of anaphylatoxins. The overactivity of the bronchioles' muscle is a result of exposure to a stimulus which under normal circumstances would cause little or no response. The resulting constriction and inflammation causes a narrowing of the airways and an increase in mucus production; this reduces the amount of oxygen that is available to the individual causing breathlessness, coughing and hypoxia.
