Horseshoe kidney pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
==Pathogenesis== | |||
The central portion of the kidney ends up inferior to the [[inferior mesenteric artery]], since its embryological ascent is arrested by its presence. This is the most common type of fusion anomaly in the kidneys. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 14:09, 27 September 2012
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Horseshoe kidney Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Horseshoe kidney pathophysiology On the Web |
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American Roentgen Ray Society Images of Horseshoe kidney pathophysiology |
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Risk calculators and risk factors for Horseshoe kidney pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathogenesis
The central portion of the kidney ends up inferior to the inferior mesenteric artery, since its embryological ascent is arrested by its presence. This is the most common type of fusion anomaly in the kidneys.