Cholesterol emboli syndrome pathophysiology

Cholesterol emboli syndrome Microchapters
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Pathophysiology

Cholesterol emboli occur when atheromatous plaques rupture and release cholesterol crystals, either spontaneously or as a result of iatrogenesis. Cholesterol emboli syndrome (CES) is defined as the occlusion of 55-900 um arterioles by cholesterol crystals after their dislodgment from eroded upstream atheromatous plaques. The occlusion site is subsequently surrounded by intimal tissue, fibrin and platelet thrombi, and foreign-body giant cells.

References

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