Renovascular disease natural history, complications and prognosis: Difference between revisions

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Revision as of 18:28, 28 September 2012

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

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 ==Prognosis==
-===Favorable Predictors===
-'''Successful Outcome For Control Of Hypertension'''
-*Rapid acceleration of hypertension over the prior weeks or months
-*Presence of “malignant” hypertension
-*Hypertension in association with flash pulmonary edema
-*Contemporaneous rise in serum creatinine
-*Development of azotemia in response to ACE inhibitors administered for control of hypertension.
-'''Successful Salvage Or Preservation Of Renal Function'''
-*Recent rapid  rise in creatinine, unexplained by other factors
-*Azotemia resulting from  ACE inhibitors
-*Absence of diabetes or other cause of intrinsic kidney disease
-*Presence of global renal ischemia, wherein the entire functioning renal mass is subtended by bilateral critically narrowed renal arteries or a vessel supplying a solitary kidney.
-===Unfavorable Predictors===
-*Renal atrophy demonstrated by kidney length <7.5 cm on ultrasound
-*High renal resistance index detected by duplex ultrasound
-*Proteinuria > 1gm/day
-*Hyperuricemia
-*Creatinine clearance <40 mL/minute
 ==References==