Oral candidiasis pathophysiology: Difference between revisions
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== Pathophysiology== | |||
Candida albicans and C. glabrata. Less commonly, C.tropicalis, C. parapsilosis, and C. krusei. Rarely, other Candida species. Most cases of OPC are caused by the person’s own Candida organisms which normally live in the mouth or digestive tract. A person has symptoms when overgrowth of Candida organisms occurs. | |||
Most infections are endogenous in origin, but organisms can be transmitted on the hands of care givers. | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Revision as of 20:19, 26 November 2012
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Oral candidiasis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Oral candidiasis pathophysiology On the Web |
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American Roentgen Ray Society Images of Oral candidiasis pathophysiology |
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Risk calculators and risk factors for Oral candidiasis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Pathophysiology
Candida albicans and C. glabrata. Less commonly, C.tropicalis, C. parapsilosis, and C. krusei. Rarely, other Candida species. Most cases of OPC are caused by the person’s own Candida organisms which normally live in the mouth or digestive tract. A person has symptoms when overgrowth of Candida organisms occurs.
Most infections are endogenous in origin, but organisms can be transmitted on the hands of care givers.