Parotitis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
Revision as of 15:13, 7 December 2012
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Parotitis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Parotitis pathophysiology On the Web |
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American Roentgen Ray Society Images of Parotitis pathophysiology |
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Risk calculators and risk factors for Parotitis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Acute infection can occur in any salivary gland but the most commonly affected one is the parotid. This is thought to be due to a combination of anatomic and physiologic factors. The saliva from the parotid is less mucoid than that from the other salivary glands. IgA, lysozyme and sialic acid are all found in smaller amounts in the more viscous parotid secretions. These substances are thought to help fight off ascending bacterial infection. Bacterial parotitis is generally unilateral in adults (75-90%), while viral is generally bilateral. Though 80-90% of salivary calculi occur in the Wharton’s duct of the submandibular gland, the parotid remains the most common site of acute suppurative salivary infection. The secretions from the submandibular gland are more alkaline, thought to result in a higher concentration of insoluble calcium phosphate.