Primary amoebic meningoencephalitis history and symptoms: Difference between revisions
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{{Primary amoebic meningoencephalitis}} | {{Primary amoebic meningoencephalitis}} | ||
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==History and Symptoms== | ==History and Symptoms== | ||
After the organisms have multiplied and largely consumed the olfactory bulbs, the infection rapidly spreads through the [[mitral cell]] [[axons]] to the rest of the [[cerebrum]], resulting in onset of frank [[encephalitis|encephalitic]] symptoms, including [[cephalgia]] (headache), [[nausea]], and [[Rigidity (neurology)|rigidity]] of the neck muscles, progressing to vomiting, [[delirium]], [[seizures]], and eventually irreversible coma. Death usually occurs within 14 days of exposure as a result of [[respiratory failure]] when the infection spreads to the [[brain stem]], destroying the autonomic nerve cells of the [[medulla oblongata]]. | After the organisms have multiplied and largely consumed the olfactory bulbs, the infection rapidly spreads through the [[mitral cell]] [[axons]] to the rest of the [[cerebrum]], resulting in onset of frank [[encephalitis|encephalitic]] symptoms, including [[cephalgia]] (headache), [[nausea]], and [[Rigidity (neurology)|rigidity]] of the neck muscles, progressing to vomiting, [[delirium]], [[seizures]], and eventually irreversible coma. Death usually occurs within 14 days of exposure as a result of [[respiratory failure]] when the infection spreads to the [[brain stem]], destroying the autonomic nerve cells of the [[medulla oblongata]]. | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Needs content]] | [[Category:Needs content]] | ||
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[[Category:Infectious disease]] | [[Category:Infectious disease]] | ||
[[Category:Neurological Disease]] | [[Category:Neurological Disease]] | ||
[[Category:Disease]] | |||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
Revision as of 21:10, 21 December 2012
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
History and Symptoms
After the organisms have multiplied and largely consumed the olfactory bulbs, the infection rapidly spreads through the mitral cell axons to the rest of the cerebrum, resulting in onset of frank encephalitic symptoms, including cephalgia (headache), nausea, and rigidity of the neck muscles, progressing to vomiting, delirium, seizures, and eventually irreversible coma. Death usually occurs within 14 days of exposure as a result of respiratory failure when the infection spreads to the brain stem, destroying the autonomic nerve cells of the medulla oblongata.