MICU intern's survival guide vasopressors: Difference between revisions
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===Norepinephrine=== | ===Norepinephrine=== | ||
* Mechanism of action - Acts on both alpha1 (vasoconstriction of blood vessels) and beta1 receptors (increases cardiac output). | * Mechanism of action - Acts on both alpha1 (vasoconstriction of blood vessels) and beta1 receptors (increases cardiac output). | ||
* | * Indications: | ||
** Septic shock | ** [[Septic shock]] | ||
** Cardiogenic shock - Recent studies have shown that norepinephrine may be better than dobutamine in cardiogenic shock due to decreased risks for tachyarrhythmias. | ** [[Cardiogenic shock]] - Recent studies have shown that [[norepinephrine]] may be better than [[dobutamine]] in cardiogenic shock due to decreased risks for tachyarrhythmias. | ||
** In cases where the type of shock is unclear norepinephrine can be tried as first line agent. | ** In cases where the type of shock is unclear norepinephrine can be tried as first line agent. | ||
** Doses - 1-30mcg/min. | ** Doses - 1-30mcg/min. | ||
Revision as of 04:02, 23 December 2012
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Norepinephrine
- Mechanism of action - Acts on both alpha1 (vasoconstriction of blood vessels) and beta1 receptors (increases cardiac output).
- Indications:
- Septic shock
- Cardiogenic shock - Recent studies have shown that norepinephrine may be better than dobutamine in cardiogenic shock due to decreased risks for tachyarrhythmias.
- In cases where the type of shock is unclear norepinephrine can be tried as first line agent.
- Doses - 1-30mcg/min.