Altitude sickness risk factors: Difference between revisions
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==Overview== | ==Overview== | ||
Individual susceptibility to high altitude pulmonary edema (HAPE) is difficult to predict. The most reliable risk factor is previous susceptibility to HAPE, and there is likely to be a genetic basis to this condition, perhaps involving the gene for [[angiotensin converting enzyme]] (ACE). | The chance of getting [[altitude sickness]] increases the faster a person climbs to a high altitude. How severe the symptoms are also depends on this factor, as well as how hard the person pushes (exerts) himself or herself. People who normally live at or near sea level are more prone to [[altitude sickness]]. Individual susceptibility to high altitude pulmonary edema (HAPE) is difficult to predict. The most reliable risk factor is previous susceptibility to HAPE, and there is likely to be a genetic basis to this condition, perhaps involving the gene for [[angiotensin converting enzyme]] (ACE). | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Revision as of 15:40, 4 February 2013
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Altitude sickness Microchapters |
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Altitude sickness risk factors On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
The chance of getting altitude sickness increases the faster a person climbs to a high altitude. How severe the symptoms are also depends on this factor, as well as how hard the person pushes (exerts) himself or herself. People who normally live at or near sea level are more prone to altitude sickness. Individual susceptibility to high altitude pulmonary edema (HAPE) is difficult to predict. The most reliable risk factor is previous susceptibility to HAPE, and there is likely to be a genetic basis to this condition, perhaps involving the gene for angiotensin converting enzyme (ACE).