Myocardial rupture overview: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. Other causes of rupture include cardiac trauma, [[endocarditis]] (infection of the heart),<ref name="Lin-2006">{{cite journal | author=Lin TH, Su HM, Voon WC, Lai HM, Yen HW, Lai WT, Sheu SH. | title=Association between hypertension and primary mitral chordae tendinae rupture. | journal=Am J Hypertens | year=2006 | volume=19 | issue=1 | pages=75-9 | id=PMID 16461195}}</ref><ref name="de Diego-2006">{{cite journal | author=de Diego C, Marcos-Alberca P, Pai RK. | title=Giant periprosthetic vegetation associated with pseudoaneurysmal-like rupture. | journal=Eur Heart J | year=2006 | volume=27 | issue=8 | pages=912 | format=PDF | url=http://eurheartj.oxfordjournals.org/cgi/reprint/27/8/912.pdf | id=PMID 16569654}}</ref> [[cardiac tumor]]s, infiltrative diseases of the heart,<ref name="Lin-2006"/> and [[aortic dissection]]. | The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. Other causes of rupture include cardiac trauma, [[endocarditis]] (infection of the heart),<ref name="Lin-2006">{{cite journal | author=Lin TH, Su HM, Voon WC, Lai HM, Yen HW, Lai WT, Sheu SH. | title=Association between hypertension and primary mitral chordae tendinae rupture. | journal=Am J Hypertens | year=2006 | volume=19 | issue=1 | pages=75-9 | id=PMID 16461195}}</ref><ref name="de Diego-2006">{{cite journal | author=de Diego C, Marcos-Alberca P, Pai RK. | title=Giant periprosthetic vegetation associated with pseudoaneurysmal-like rupture. | journal=Eur Heart J | year=2006 | volume=27 | issue=8 | pages=912 | format=PDF | url=http://eurheartj.oxfordjournals.org/cgi/reprint/27/8/912.pdf | id=PMID 16569654}}</ref> [[cardiac tumor]]s, infiltrative diseases of the heart,<ref name="Lin-2006"/> and [[aortic dissection]]. | ||
==Risk Factors== | |||
Risk factors for rupture after an acute myocardial infarction include female gender, advanced age of the individual, and a low [[body mass index]]. Other presenting signs associated with myocardial rupture include a pericardial friction rub, sluggish flow in the coronary artery after it is opened, the [[left anterior descending artery]] being the cause of the acute MI,<ref name="Sugiura-2003">{{cite journal | author=Sugiura T, Nagahama Y, Nakamura S, Kudo Y, Yamasaki F, Iwasaka T. | title=Left ventricular free wall rupture after reperfusion therapy for acute myocardial infarction. | journal=Am J Cardiol | year=2003 | volume=92 | issue=3 | pages=282-4 | id=PMID 12888132}}</ref> and delay of revascularization greater than 2 hours. | |||
==References== | ==References== | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]
Overview
Myocardial rupture is a laceration or tearing of the walls of the ventricles or atria of the heart, of the interatrial or interventricular septum, of the papillary muscles or chordae tendineae or of one of the valves of the heart. It is most commonly seen as a serious sequelae of an acute myocardial infarction (heart attack).
Pathophysiology
The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. Other causes of rupture include cardiac trauma, endocarditis (infection of the heart),[1][2] cardiac tumors, infiltrative diseases of the heart,[1] and aortic dissection.
Risk Factors
Risk factors for rupture after an acute myocardial infarction include female gender, advanced age of the individual, and a low body mass index. Other presenting signs associated with myocardial rupture include a pericardial friction rub, sluggish flow in the coronary artery after it is opened, the left anterior descending artery being the cause of the acute MI,[3] and delay of revascularization greater than 2 hours.
References
- ↑ 1.0 1.1 Lin TH, Su HM, Voon WC, Lai HM, Yen HW, Lai WT, Sheu SH. (2006). "Association between hypertension and primary mitral chordae tendinae rupture". Am J Hypertens. 19 (1): 75–9. PMID 16461195.
- ↑ de Diego C, Marcos-Alberca P, Pai RK. (2006). "Giant periprosthetic vegetation associated with pseudoaneurysmal-like rupture" (PDF). Eur Heart J. 27 (8): 912. PMID 16569654.
- ↑ Sugiura T, Nagahama Y, Nakamura S, Kudo Y, Yamasaki F, Iwasaka T. (2003). "Left ventricular free wall rupture after reperfusion therapy for acute myocardial infarction". Am J Cardiol. 92 (3): 282–4. PMID 12888132.