Total anomalous pulmonary venous connection overview: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
In patients of Total anomalous pulmonary venous connection (TAPVC) there is a mixing of oxygenated pulmonary venous blood with deoxygenated blood from systemic circulation. | In patients of Total anomalous pulmonary venous connection (TAPVC) there is a mixing of oxygenated pulmonary venous blood with deoxygenated blood from systemic circulation. | ||
==Causes== | |||
Like many other congenital heart defects, the exact cause of total anomalous pulmonary venous connection is not known. Few factors like genetics, maternal alcohol syndrome and some drugs have been found to occur with increased frequency. | |||
==References== | ==References== |
Revision as of 19:00, 5 March 2013
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Associate Editor-In-Chief:Cafer Zorkun, M.D., Ph.D. [2]; Keri Shafer, M.D. [3] Priyamvada Singh, MBBS [[4]] Assistant Editor-In-Chief: Kristin Feeney, B.S. [[5]]
Overview
Total anomalous pulmonary venous connection (TAPVC) is a rare cyanotic congenital heart defect (CHD) in which all four pulmonary veins are malpositioned and make anomalous connections to the systemic venous circulation. (Normally, pulmonary venous return carries oxygenated blood to the left atrium and to the rest of the body). A patent foramen ovale or an atrial septal defect must be present in order to allow systemic blood flow.
Pathophysiology
In patients of Total anomalous pulmonary venous connection (TAPVC) there is a mixing of oxygenated pulmonary venous blood with deoxygenated blood from systemic circulation.
Causes
Like many other congenital heart defects, the exact cause of total anomalous pulmonary venous connection is not known. Few factors like genetics, maternal alcohol syndrome and some drugs have been found to occur with increased frequency.