Portal hypertension: Difference between revisions

	Portal Hypertension Microchapters
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Overview
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Pathophysiology
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Differentiating Portal Hypertension from other Diseases
Epidemiology and Demographics
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	Portal hypertension;
	The portal vein and its tributaries.

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Revision as of 15:20, 16 April 2013

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1],Assistant Editor-in-Chief: Soumya Sachdeva

Pathophysiology

Natural History and Complications

Diagnosis

Treatment

Medical management

Percutaneous interventions

Transjugular intrahepatic portosystemic shunting is the creation of a connection between the portal and the venous system. As the pressure over the venous system is lower than over a hypertensive portal system, this would decrease the pressure over the portal system and a decreased risk of complications.

Surgical interventions

The most definitive treatment of portal hypertension is a liver transplant.

References

External links

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 ==Treatment==
 ===Medical management===
-Treatment with a non-selective [[beta blocker]] is often commenced once [[portal hypertension]] has been diagnosed, and almost always if there has already been bleeding from esophageal varices. Typically, this is done with either [[propranolol]] or [[nadolol]]. The addition of a [[nitrate]], such as [[isosorbide mononitrate]], to the [[beta blocker]] is more effective than using beta blockers alone and may be the preferred regimen in those people with portal [[hypertension]] who have already experienced variceal bleeding. In acute or severe complications of the [[hypertension]], such as bleeding varices, intravenous [[octreotide]] (a [[somatostatin]] analogue) or intravenous [[terlipressin]] (an [[antidiuretic hormone]] analogue) is commenced to decrease the portal pressure.
 ===Percutaneous interventions===