Renal artery stenosis resident survival guide: Difference between revisions

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<tr class="v-firstrow"><th>Scenario</th><th>Level of evidence</th></tr>
<tr class="v-firstrow"><th>Scenario</th><th>Level of evidence</th></tr>
<tr><td>'''1.'''Onset of hypertension before the age of 30 years or severe hypertension after the age of 55</td><td>Class I; LOE B</td></tr>
<tr><td>'''1.'''Onset of hypertension before the age of 30 years or severe hypertension after the age of 55</td><td>Class I; LOE B</td></tr>
<tr><td>2. Accelerated, resistant, or malignant hypertension</td><td>Class I; LOE C</td></tr>
<tr><td>'''2.''' Accelerated, resistant, or malignant hypertension</td><td>Class I; LOE C</td></tr>
<tr><td>3. Development of new azotemia or worsening renal function after administration of an ACE inhibitor or ARB agent</td><td>Class I; LOE B</td></tr>
<tr><td>'''3.''' Development of new azotemia or worsening renal function after administration of an ACE inhibitor or ARB agent</td><td>Class I; LOE B</td></tr>
<tr><td>4. Unexplained atrophic kidney or size discrepancy between kidneys of greater than 1.5 cm</td><td>Class I; LOE B</td></tr>
<tr><td>'''4.''' Unexplained atrophic kidney or size discrepancy between kidneys of greater than 1.5 cm</td><td>Class I; LOE B</td></tr>
<tr><td>5. Sudden, unexplained pulmonary edema</td><td>Class I; LOE B</td></tr>
<tr><td>'''5.''' Sudden, unexplained pulmonary edema</td><td>Class I; LOE B</td></tr>
<tr><td>6. Unexplained renal dysfunction, including individuals starting renal replacement therapy</td><td>Class IIa; LOE B</td></tr>
<tr><td>'''6.''' Unexplained renal dysfunction, including individuals starting renal replacement therapy</td><td>Class IIa; LOE B</td></tr>
<tr><td>7. Multi-vessel coronary artery disease</td><td>Class IIb; LOE B</td></tr>
<tr><td>'''7.''' Multi-vessel coronary artery disease</td><td>Class IIb; LOE B</td></tr>
<tr><td>8. Unexplained congestive heart failure</td><td>Class IIb; LOE C</td></tr>
<tr><td>'''8.''' Unexplained congestive heart failure</td><td>Class IIb; LOE C</td></tr>
<tr><td>9. Refractory angina</td><td>Class IIb; LOE C</td></tr>
<tr><td>'''9.''' Refractory angina</td><td>Class IIb; LOE C</td></tr>
</table>}}
</table>}}
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{{familytree | B01 | | B02 | B01=<div style="height: 1em; width: 25em; padding:1em;">'''Noninvasive Imaging'''
{{familytree | B01 | | B02 | B01=<div style="height: 1em; width: 25em; padding:1em;">'''Noninvasive Imaging'''  
*Duplex ultrasound
*Gadolinium enhanced MRA
*CT angiography'''  
</div>| B02= <div style="height: 1em; width: 25em; padding:1em;">'''Invasive Imaging'''
</div>| B02= <div style="height: 1em; width: 25em; padding:1em;">'''Invasive Imaging'''
*Abdominal aortography to assess the renal arteries during coronary and peripheral angiography</div>}}
</div>}}


{{familytree | |!| | | |!| | }}
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{{familytree | C01 | | C02 | C01= <div style="float: left; text-align: left; height: 3em; width: 25em; padding:1em;">❑ Do not monitor platelet count </div>| C02= <div style="float: left; text-align: left; height: 3em; width: 25em; padding:1em;">❑ Monitor platelet count every 2 or 3 days from day 4 to day 14 (or until heparin is stopped) </div>}}
{{familytree | C01 | | C02 | C01= <div style="float: left; text-align: left; height: 3em; width: 25em; padding:1em;">❑ Duplex ultrasound
❑ Gadolinium enhanced MRA
❑ CT angiography </div>| C02= <div style="float: left; text-align: left; height: 3em; width: 25em; padding:1em;">❑ Abdominal aortography to assess the renal arteries during coronary and peripheral angiography </div>}}


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{{familytree/end}}

Revision as of 06:09, 6 January 2014

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Karol Gema Hernandez, M.D. [2]

Definition

This section provides a short and straight to the point definition of the disease or symptom in one sentence.

Causes

Life Threatening Causes

Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.

Common Causes

Clinical Clues to the Diagnosis of RAS

 
 
Determine if one or more of the above is present
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
ScenarioLevel of evidence
1.Onset of hypertension before the age of 30 years or severe hypertension after the age of 55Class I; LOE B
2. Accelerated, resistant, or malignant hypertensionClass I; LOE C
3. Development of new azotemia or worsening renal function after administration of an ACE inhibitor or ARB agentClass I; LOE B
4. Unexplained atrophic kidney or size discrepancy between kidneys of greater than 1.5 cmClass I; LOE B
5. Sudden, unexplained pulmonary edemaClass I; LOE B
6. Unexplained renal dysfunction, including individuals starting renal replacement therapyClass IIa; LOE B
7. Multi-vessel coronary artery diseaseClass IIb; LOE B
8. Unexplained congestive heart failureClass IIb; LOE C
9. Refractory anginaClass IIb; LOE C
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Proceed to further diagnostic testing
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Noninvasive Imaging
 
Invasive Imaging
 
 
 
 
 
 
 
 
 
 
❑ Duplex ultrasound

❑ Gadolinium enhanced MRA

❑ CT angiography
 
❑ Abdominal aortography to assess the renal arteries during coronary and peripheral angiography

Algorithm based on the 2012 ACCP evidence based clinical practice guidelines.[1]



References


Template:WikiDoc Sources

  1. Linkins LA, Dans AL, Moores LK, Bona R, Davidson BL, Schulman S; et al. (2012). "Treatment and prevention of heparin-induced thrombocytopenia: Antithrombotic Therapy and Prevention of Thrombosis, 9th ed: American College of Chest Physicians Evidence-Based Clinical Practice Guidelines". Chest. 141 (2 Suppl): e495S–530S. doi:10.1378/chest.11-2303. PMC 3278058. PMID 22315270.