Sulfadiazine: Difference between revisions
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==Mechanism of Action== | ==Mechanism of Action== | ||
The systemic sulfonamides are bacteriostatic agents having a similar spectrum of activity. Sulfonamides competitively inhibit bacterial synthesis of folic acid (pteroylglutamic acid) from aminobenzoic acid. Resistant strains are capable of utilizing folic acid precursors or preformed folic acid.<ref name="dailymed.nlm.nih.gov">{{Cite web | last = | first = | title = SULFADIAZINE TABLET [EON LABS, INC.] | url = http://dailymed.nlm.nih.gov/dailymed/lookup.cfm?setid=10549cba-9c15-4d2e-a68c-5afbc178591d | publisher = | date = | accessdate}}</ref> | |||
==References== | ==References== |
Revision as of 08:20, 9 January 2014
For patient information, click here.
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mohamed Moubarak, M.D. [2]
Overview
Category
US Brand Names
SULFADIAZINE®
FDA Package Insert
Description | Clinical Pharmacology | Microbiology | Indications and Usage | Contraindications | Warnings and Precautions | Adverse Reactions | Overdosage | Dosage and Administration | How Supplied | Labels and Packages
Mechanism of Action
The systemic sulfonamides are bacteriostatic agents having a similar spectrum of activity. Sulfonamides competitively inhibit bacterial synthesis of folic acid (pteroylglutamic acid) from aminobenzoic acid. Resistant strains are capable of utilizing folic acid precursors or preformed folic acid.[1]
References
- ↑ "SULFADIAZINE TABLET [EON LABS, INC.]". Text " accessdate" ignored (help)