Cardiac tamponade resident survival guide: Difference between revisions
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❑ '''Obtain a detailed history:'''<br>♦ Time course of illness<br>♦ Concurrent medical illness - hy<br>♦ History of chest infection<br> | |||
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❑ '''Examine the patient:'''<br>♦ Head/Neck - Neck veins (flat, no ↑JVP)<br>♦ Chest - No S3/S4, no murmurs<br>♦ Limbs - Hyperdynamic pulses, no edema</div>}} | |||
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Revision as of 21:53, 27 January 2014
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Karol Gema Hernandez, M.D. [2] Ayokunle Olubaniyi, M.B,B.S [3]
Definition
This is a medical emergency which involves a slow or rapid compression of the heart due to the accumulation of fluid, pus, blood, clots, or gas in the pericardium, as a result of effusion, trauma, or rupture of the heart.[1] It is suspected based on history, physical examination, and chest imaging findings, but it is confirmed based on the clinical response observed during or post-pericardial fluid drainage.
Causes
Life Threatening Causes
Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.
Common Causes
Acute Cardiac Tamponade
- Ascending aortic dissection
- Iatrogenic - central line insertion, pacemaker insertion, coronary interventions, myocardial biopsy
- Penetrating trauma
- Post myocardial infarction treatments - heparin, thrombolytics
Subacute Cardiac Tamponade
- Collagen vascular diseases
- Idiopathic
- Malignancy - breast, Kaposi's sarcoma, lung, lymphomas
- Pericarditis
- Viral - coxsachie, echo, CMV
- Bacterial - pneumococcus, streptococcus, staphylococcus
- Fungal - blastomyces, cryptococcus, histoplasma, pneumocystis carinii
- Tuberculous
Management
Characterize the symptoms:
Or as a feature of a complication
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Patient evaluation:
❑ Obtain a detailed history: ❑ Examine the patient: ♦ Head/Neck - Neck veins (flat, no ↑JVP) ♦ Chest - No S3/S4, no murmurs ♦ Limbs - Hyperdynamic pulses, no edema | |||||||||||||||||||||||||
Dos
- Always suspect cardiac tamponade in any patient presenting with hypotension, tachycardia and distended neck veins (or elevated jugular venous pressure).
- Always measure pulsus paradoxus whenever you suspect cardiac tamponade.
- Consider the intrapericardial pressure more than the volume of the pericardial fluid in the management of cardiac tamponade.
- The drainage of pericardial effusion should be gradual and slow to avoid the precipitation of pulmonary edema.
- Echocardiography is the primary modality of choice, considering its high specificity and sensitivity, low cost and lack of radiation. CT and cardiac MRI are indicated when echocardiography is inconclusive.
- Consider 2D and doppler echocardiography prior to discharge to confirm total removal or detect reaccumulation of pericardial fluid.
Don'ts
- Never delay treatment whenever you suspect cardiac tamponade.
- Avoid diuretics because it may worsen the central venous pressure.
- Avoid placing pericardial fluid drainage catheter in situ for > 3 days.
- Avoid the subcostal approach of pericardiocentesis if coagulopathy is present. Iatrogenic injuries to the liver may be life-threatening.
- Avoid positive pressure mechanical ventilation. It may further reduce cardiac filling.
- The use of inotropic agents for hemodynamic support should not be a substitute or cause a delay to pericadiocentesis.