Cardiac tamponade resident survival guide: Difference between revisions

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{{familytree | | | B01 | |B01=<div style="float: left; text-align: left; height: 14em; width: 40em; padding:1em;">'''Patient evaluation:'''
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❑ '''Obtain a detailed history:'''<br>♦ Time course of illness<br>♦ Concurrent medical illness - hy<br>♦ History of chest infection<br>
❑ '''Obtain a detailed history:'''<br>♦ Time course of illness<br>♦ Concurrent medical illness - [[hypothyroidism]], [[systemic lupus erythematosus]]<br>♦ Trauma
<br>♦ Radiation therapy<br>♦ Recent cardiac therapeutic procedures<br>♦ Recent [[myocardial infarction]]
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❑ '''Examine the patient:'''<br>♦ Head/Neck - Neck veins (flat, no ↑JVP)<br>♦ Chest - No S3/S4, no murmurs<br>♦ Limbs - Hyperdynamic pulses, no edema</div>}}
❑ '''Examine the patient:'''<br>❑ General- distended neck veins (↑JVP)<br>❑ Vitals <br>♦ [[Tachycardia]], [[tachypnea]], [[hypotension]]<br>❑ Cardiopulmonary<br>♦ Clear lungs<br>♦ Distant (muffled) heart sounds<br>♦ Pericardial friction rub<br>❑ Limbs - peripheral edema <br> '''Note''' <br>♦ Measure BP and response to inspiration ([[pulsus paradoxus]])<br>♦ [[Bradycardia]] (especially in [[uremia]] or [[hypothyroidism]]</div>}}
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Revision as of 13:17, 28 January 2014

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Karol Gema Hernandez, M.D. [2] Ayokunle Olubaniyi, M.B,B.S [3]

Definition

This is a medical emergency which involves a slow or rapid compression of the heart due to the accumulation of fluid, pus, blood, clots, or gas in the pericardium, as a result of effusion, trauma, or rupture of the heart.[1] It is suspected based on history, physical examination, and chest imaging findings, but it is confirmed based on the clinical response observed during or post-pericardial fluid drainage.

Causes

Life Threatening Causes

Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.

Common Causes

Acute Cardiac Tamponade

Subacute Cardiac Tamponade

Viral - coxsachie, echo, CMV
Bacterial - pneumococcus, streptococcus, staphylococcus
Fungal - blastomyces, cryptococcus, histoplasma, pneumocystis carinii
Tuberculous

Management

 
 
Characterize the symptoms:
❑ Chest pain❑ Cough
❑ Cyanosis ❑ Dysphagia
❑ Dyspnea ❑ Fatigue
❑ Fever❑ Near syncope
❑ Orthopnea❑ Peripheral edema

Or as a feature of a complication

❑ Renal failure❑ Abdominal plethora
❑ Shock liver ❑ Mesenteric ischemia
 
 
 
 
 
 
 
 
 
 
Patient evaluation:

Obtain a detailed history:
♦ Time course of illness
♦ Concurrent medical illness - hypothyroidism, systemic lupus erythematosus
♦ Trauma
♦ Radiation therapy
♦ Recent cardiac therapeutic procedures
♦ Recent myocardial infarction


Examine the patient:
❑ General- distended neck veins (↑JVP)
❑ Vitals
Tachycardia, tachypnea, hypotension
❑ Cardiopulmonary
♦ Clear lungs
♦ Distant (muffled) heart sounds
♦ Pericardial friction rub
❑ Limbs - peripheral edema
Note
♦ Measure BP and response to inspiration (pulsus paradoxus)
Bradycardia (especially in uremia or hypothyroidism
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 

Dos

  • Always suspect cardiac tamponade in any patient presenting with hypotension, tachycardia and distended neck veins (or elevated jugular venous pressure).
  • Always measure pulsus paradoxus whenever you suspect cardiac tamponade.
  • Consider the intrapericardial pressure more than the volume of the pericardial fluid in the management of cardiac tamponade.
  • The drainage of pericardial effusion should be gradual and slow to avoid the precipitation of pulmonary edema.
  • Echocardiography is the primary modality of choice, considering its high specificity and sensitivity, low cost and lack of radiation. CT and cardiac MRI are indicated when echocardiography is inconclusive.
  • Consider 2D and doppler echocardiography prior to discharge to confirm total removal or detect reaccumulation of pericardial fluid.

Don'ts

  • Never delay treatment whenever you suspect cardiac tamponade.
  • Avoid diuretics because it may worsen the central venous pressure.
  • Avoid placing pericardial fluid drainage catheter in situ for > 3 days.
  • Avoid the subcostal approach of pericardiocentesis if coagulopathy is present. Iatrogenic injuries to the liver may be life-threatening.
  • Avoid positive pressure mechanical ventilation. It may further reduce cardiac filling.
  • The use of inotropic agents for hemodynamic support should not be a substitute or cause a delay to pericadiocentesis.

References

  1. Spodick, DH. (2003). "Acute cardiac tamponade". N Engl J Med. 349 (7): 684–90. doi:10.1056/NEJMra022643. PMID 12917306. Unknown parameter |month= ignored (help)

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