Right ventricular myocardial infarction echocardiography or ultrasound: Difference between revisions

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*** Where RAP equals the estimated right atrial pressure based upon examination of the jugular neck veins, V is the velocity of the [[tricuspid insufficiency]] jet by Doppler ultrasonography, and V<sup>2</sup> refers to V squared. A right ventricular peak systolic pressure above 30 mmHg is considered elevated. Mild elevation is 30 to 45 mmHg, moderate 45 to 60 mmHg, and severe is greater than 60 mmHg.
*** Where RAP equals the estimated right atrial pressure based upon examination of the jugular neck veins, V is the velocity of the [[tricuspid insufficiency]] jet by Doppler ultrasonography, and V<sup>2</sup> refers to V squared. A right ventricular peak systolic pressure above 30 mmHg is considered elevated. Mild elevation is 30 to 45 mmHg, moderate 45 to 60 mmHg, and severe is greater than 60 mmHg.
* In the absence of [[pulmonary stenosis]] (which is rare), right ventricular systolic pressure is equal to the pulmonary artery systolic pressure. As a result, this equation is useful for estimating the presence and severity of [[pulmonary hypertension]]. When significant [[pulmonary artery hypertension]] (> 45 to 50 mmHg) complicates [[Right ventricular myocardial infarction|right ventricular infarction]], the failing right ventricle may be unable to pump blood from the right heart into the left heart. In this setting, a positive inotropic agent such as [[dobutamine]] may be effective in augmenting forward flow while also decreasing [[pulmonary vascular resistance]] and right ventricular overload.
* In the absence of [[pulmonary stenosis]] (which is rare), right ventricular systolic pressure is equal to the pulmonary artery systolic pressure. As a result, this equation is useful for estimating the presence and severity of [[pulmonary hypertension]]. When significant [[pulmonary artery hypertension]] (> 45 to 50 mmHg) complicates [[Right ventricular myocardial infarction|right ventricular infarction]], the failing right ventricle may be unable to pump blood from the right heart into the left heart. In this setting, a positive inotropic agent such as [[dobutamine]] may be effective in augmenting forward flow while also decreasing [[pulmonary vascular resistance]] and right ventricular overload.
Shown below are an animated image and a static image depicting TIMI flow grade 0 in the [[RCA]].  Outlined in blue in the second image is the flow of the dye which completely stops at the level of the stenosis.  Outlined with a yellow dashed line is the pathway that the dye would have filled in case the stenosis was absent.
[[Image:TFG-0-RCA.gif|300px|TIMI flow grade 0 in RCA]]
[[Image:TFG0RCA-static.gif|300px|TIMI flow grade 0 in RCA: Outlined in blue in the second image is the flow of the dye which completely stops at the level of the stenosis; outlined with a yellow dashed line is the pathway that the dye would have filled in case the stenosis was absent.]]


==References==
==References==

Revision as of 22:43, 11 March 2014

Right ventricular myocardial infarction Microchapters

Home

Overview

Pathophysiology

Pathophysiology of Reperfusion
Gross Pathology
Histopathology

Causes

Differentiating Right ventricular myocardial infarction from other Diseases

Epidemiology and Demographics

Risk Factors

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Criteria

History and Symptoms

Physical Examination

Electrocardiogram

Chest X Ray

Echocardiography or Ultrasound

Coronary Angiography

Other Imaging Findings

Treatment

Initial Care

Pharmacological Reperfusion

Reperfusion Therapy (Overview of Fibrinolysis and Primary PCI)
Fibrinolysis

Mechanical Reperfusion

The Importance of Reducing Door-to-Balloon Times
Primary PCI
Adjunctive and Rescue PCI
Rescue PCI
Facilitated PCI
Adjunctive PCI
CABG
Management of Patients Who Were Not Reperfused
Assessing Success of Reperfusion

Antithrombin Therapy

Antithrombin Therapy
Unfractionated Heparin
Low Molecular Weight Heparinoid Therapy
Direct Thrombin Inhibitor Therapy
Factor Xa Inhibition
DVT Prophylaxis
Long Term Anticoagulation

Antiplatelet Agents

Aspirin
Thienopyridine Therapy
Glycoprotein IIbIIIa Inhibition

Other Initial Therapy

Inhibition of the Renin-Angiotensin-Aldosterone System
Magnesium Therapy
Glucose Control
Calcium Channel Blocker Therapy

Right ventricular myocardial infarction echocardiography or ultrasound On the Web

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US National Guidelines Clearinghouse

NICE Guidance

FDA on Right ventricular myocardial infarction echocardiography or ultrasound

CDC on Right ventricular myocardial infarction echocardiography or ultrasound

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Directions to Hospitals Treating Right ventricular myocardial infarction

Risk calculators and risk factors for Right ventricular myocardial infarction echocardiography or ultrasound

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Overview

Echocardiography

  • Echocardiography may be limited in right ventricular infarction by suboptimal views of the right ventricle. In addition, interpretation of right ventricular function may be affected by coexistent pulmonary disease (such as obstructive lung disease or pulmonary embolism).
  • Despite these limitations, echocardiography is often a useful test that can be performed at the bedside when the diagnosis of right ventricular infarction is suspected. Right ventricular size, function, and the degree (if any) of tricuspid insufficiency can all be evaluated. [1] Useful information concerning left-sided structures and function can also be obtained. [2][3]
  • Right ventricular peak systolic pressure may be estimated from the Doppler signal of tricuspid insufficiency using the modified Bernoulli equation:
    • Right ventricular peak systolic pressure = RAP + 4V2
      • Where RAP equals the estimated right atrial pressure based upon examination of the jugular neck veins, V is the velocity of the tricuspid insufficiency jet by Doppler ultrasonography, and V2 refers to V squared. A right ventricular peak systolic pressure above 30 mmHg is considered elevated. Mild elevation is 30 to 45 mmHg, moderate 45 to 60 mmHg, and severe is greater than 60 mmHg.
  • In the absence of pulmonary stenosis (which is rare), right ventricular systolic pressure is equal to the pulmonary artery systolic pressure. As a result, this equation is useful for estimating the presence and severity of pulmonary hypertension. When significant pulmonary artery hypertension (> 45 to 50 mmHg) complicates right ventricular infarction, the failing right ventricle may be unable to pump blood from the right heart into the left heart. In this setting, a positive inotropic agent such as dobutamine may be effective in augmenting forward flow while also decreasing pulmonary vascular resistance and right ventricular overload.

Shown below are an animated image and a static image depicting TIMI flow grade 0 in the RCA. Outlined in blue in the second image is the flow of the dye which completely stops at the level of the stenosis. Outlined with a yellow dashed line is the pathway that the dye would have filled in case the stenosis was absent.

TIMI flow grade 0 in RCA TIMI flow grade 0 in RCA: Outlined in blue in the second image is the flow of the dye which completely stops at the level of the stenosis; outlined with a yellow dashed line is the pathway that the dye would have filled in case the stenosis was absent.

References

  1. Zehender, M, Kasper, W, Kauder, E, et al. Right ventricular infarction as an independent predictor of prognosis after acute inferior myocardial infarction. N Engl J Med 1993; 328:981. PMID 8450875
  2. Zehender, M, Kasper, W, Kauder, E, et al. Right ventricular infarction as an independent predictor of prognosis after acute inferior myocardial infarction. N Engl J Med 1993; 328:981. PMID 8450875
  3. Kahn, JK, Bernstein, M, Bengston, JR. Isolated right ventricular myocardial infarction. Ann Intern Med 1993; 118:708. PMID 8460858

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