Anhedonia pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
Anhedonia is being studied with variety of neuropsychiatrie disorders. Behavioral, electrophysiological, hemodynamic, and interview-based measures, but the most interesting findings concern neuropharmacological and neuroanatomical studies. The prevalent hypothesis is that the dopamine plays an important role in pathogenesis of anhedonia, anatomically there is a restricted activity of ventral striatum, including the nucleus accumbens and increased activation of ventral region of the prefrontal cortex, including the ventromedial prefrontal cortex and the orbitofrontal cortex. | |||
==Pathophysiology== | |||
<ref>{{Cite web | last = | first = | title = Neurobiological mechanisms of anhedonia | url = http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3181880/ | publisher = | date = | accessdate = }}</ref> | <ref>{{Cite web | last = | first = | title = Neurobiological mechanisms of anhedonia | url = http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3181880/ | publisher = | date = | accessdate = }}</ref> | ||
Revision as of 23:06, 13 March 2014
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Pratik Bahekar, MBBS [2]
Overview
Anhedonia is being studied with variety of neuropsychiatrie disorders. Behavioral, electrophysiological, hemodynamic, and interview-based measures, but the most interesting findings concern neuropharmacological and neuroanatomical studies. The prevalent hypothesis is that the dopamine plays an important role in pathogenesis of anhedonia, anatomically there is a restricted activity of ventral striatum, including the nucleus accumbens and increased activation of ventral region of the prefrontal cortex, including the ventromedial prefrontal cortex and the orbitofrontal cortex.