Sandbox chetan: Difference between revisions
Line 32: | Line 32: | ||
:❑ Majority of the patients are asymptomatic (Detected incidentally)<br> | :❑ Majority of the patients are asymptomatic (Detected incidentally)<br> | ||
❑ Symptomatic but not ruptured | ❑ Symptomatic but not ruptured | ||
:❑ Pain | :❑ Pain with an indolent onset <br> | ||
::❑ Abdominal pain | ::❑ Abdominal pain | ||
::❑ Back pain | ::❑ Back pain | ||
Line 42: | Line 42: | ||
::❑ Malaise | ::❑ Malaise | ||
❑ Symptomatic and ruptured | ❑ Symptomatic and ruptured | ||
:❑ Severe pain | :❑ Severe pain described as severe, sudden, persistent, or constant | ||
:: | |||
:❑ Hypotension | :❑ Hypotension | ||
:❑ [[Syncope]], [[loss of consciousness|fainting]] (suggestive of [[hemorrhage]])<br> | :❑ [[Syncope]], [[loss of consciousness|fainting]] (suggestive of [[hemorrhage]])<br> | ||
Line 54: | Line 55: | ||
{{familytree | | | | | | | | | |!| | | | | | | | | | | | |}} | {{familytree | | | | | | | | | |!| | | | | | | | | | | | |}} | ||
{{familytree | | | | | | | | | B01 | | | | | | | | | | | | |B01=<div style="float: left; text-align: left; padding:1em;"> Obtain a detailed history: | {{familytree | | | | | | | | | B01 | | | | | | | | | | | | |B01=<div style="float: left; text-align: left; padding:1em;"> Obtain a detailed history: | ||
❑ History to find out the risk factors for development of aneurysm | ❑ History to find out the risk factors for development of aneurysm | ||
:❑ Hyperlipidemia | :❑ Hyperlipidemia | ||
Line 62: | Line 62: | ||
::❑ Relapsing polychondritis | ::❑ Relapsing polychondritis | ||
::❑ Pseudoxanthoma elasticum | ::❑ Pseudoxanthoma elasticum | ||
::❑ Polycystic kidney disease | |||
::❑ Loeys-Dietz syndrome | |||
:❑ COPD (Emphysema) | :❑ COPD (Emphysema) | ||
:❑ Hypertension | :❑ Hypertension | ||
Line 72: | Line 74: | ||
:❑ Cardiac or renal transplant | :❑ Cardiac or renal transplant | ||
:❑ Hypertension | :❑ Hypertension | ||
❑ Family history<ref>{{cite journal |author=Clifton MA |title=Familial abdominal aortic aneurysms |journal=Br J Surg. |volume=64 |issue=11 |pages=765–6 |date=Nov 1977 |pmid=588966|doi=10.1002/bjs.1800641102 }}</ref> | ❑ Family history<ref>{{cite journal |author=Clifton MA |title=Familial abdominal aortic aneurysms |journal=Br J Surg. |volume=64 |issue=11 |pages=765–6 |date=Nov 1977 |pmid=588966|doi=10.1002/bjs.1800641102 }}</ref> | ||
:❑ Abdominal aortic aneurysm | |||
:❑ Alpha 1-antitrypsin deficiency | :❑ Alpha 1-antitrypsin deficiency | ||
❑ Past Medical History | ❑ Past Medical History | ||
Line 78: | Line 81: | ||
:❑ Atherosclerosis<br> | :❑ Atherosclerosis<br> | ||
:❑ Peripheral artery disease<br> | :❑ Peripheral artery disease<br> | ||
:❑ Giant cell arteritis<ref name="Josselin-Mahr-2013">{{Cite journal | last1 = Josselin-Mahr | first1 = L. | last2 = El Hessen | first2 = TA. | last3 = Toledano | first3 = C. | last4 = Fardet | first4 = L. | last5 = Kettaneh | first5 = A. | last6 = Tiev | first6 = K. | last7 = Cabane | first7 = J. | title = [Inflammatory aortitis in giant cell arteritis]. | journal = Presse Med | volume = 42 | issue = 2 | pages = 151-9 | month = Feb | year = 2013 | doi = 10.1016/j.lpm.2012.03.003 | PMID = 22552044 }}</ref> <br> | |||
:❑ Hemorrhoids<br> | :❑ Hemorrhoids<br> | ||
:❑ Esophageal varices<br>❑ | :❑ Esophageal varices<br> | ||
❑ Social History | |||
:❑ Smoking History (Strongest risk factor) (smoked at some point in their life)<ref name="Greenhalgh RM, Powell JT 2008 494–501">{{cite journal |author=Greenhalgh RM, Powell JT |title=Endovascular repair of abdominal aortic aneurysm |journal=N. Engl. J. Med. |volume=358 |issue=5 |pages=494–501 |date= |pmid=18234753 |doi=10.1056/NEJMct0707524 }}</ref> <br> | |||
:❑ Alcohol History<br> | :❑ Alcohol History<br> | ||
❑ Anatomic deformities | |||
❑ | :❑ Bicuspid aortic valve | ||
:❑ Coarctation of the aorta | |||
❑ Infections of the aorta (aortitis)(very rare)<br> | |||
:❑ Syphilis | |||
:❑ Salmonella | |||
:❑ Staphylococcus | |||
❑ Trauma <br> | ❑ Trauma <br> | ||
❑ Arteritis <br> | ❑ Arteritis <br> | ||
❑ Cystic medial necrosis </div>}} | ❑ Cystic medial necrosis </div>}} | ||
{{familytree | | | | | | | | | |!| | | | | | | | | | | | |}} | |||
{{familytree | | | | | | | | | C01 | | | | | | | | | | | |C01= <div style="float: left; text-align: left; padding:1em;"> Examine the patient: <br> | |||
{{familytree | | | | | | |,|-|-|^|-|-|.| | | | | | | | | | |}} | {{familytree | | | | | | |,|-|-|^|-|-|.| | | | | | | | | | |}} | ||
{{familytree | | | | | | B01 | | | | B02 | | | | | | | | | | |}} | {{familytree | | | | | | B01 | | | | B02 | | | | | | | | | | |}} |
Revision as of 15:22, 1 May 2014
Overview
An abdominal aortic aneurysm is a localized dilatation of the abdominal aorta, that exceeds the normal diameter of the abdominal aorta by more than 50%. The normal diameter of an aorta depends on the patient's age, sex, height, weight, race, body surface area, and baseline blood pressure. On average, the normal diameter of the infrarenal aorta is 2 cm, and therefore a true AAA measures 3.0 cm or more. Aortic ectasia is a mild generalized dilatation (<50% of the normal diameter of ≤ 2.9 cm) that is due to age-related degenerative changes in the vessel walls.
Causes
Life Threatening Causes
Abdominal aortic aneurysm is a life-threatening condition and must be treated as such irrespective of the underlying cause.
Common Causes
FIRE: Focused Initial Rapid Evaluation
A Focused Initial Rapid Evaluation (FIRE) should be performed to identify patients in need of immediate intervention.
Abbreviations:
Complete Diagnostic Approach
A complete diagnostic approach should be carried out after a focused initial rapid evaluation is conducted and following initiation of any urgent intervention.
{{familytree | | | | | | | | | C01 | | | | | | | | | | | |C01= Characterize the symptoms:
❑ Asymptomatic
❑ Symptomatic but not ruptured
❑ Symptomatic and ruptured
| |||||||||||||||||||||||||||||||||||||||||||||||||
Obtain a detailed history:
❑ History to find out the risk factors for development of aneurysm
❑ History to find out the risk factors for expansion of aneurysm
❑ History to find out the risk factors for rupture of aneurysm
❑ Family history[4]
❑ Past Medical History
❑ Social History
❑ Anatomic deformities
❑ Infections of the aorta (aortitis)(very rare)
❑ Trauma | |||||||||||||||||||||||||||||||||||||||||||||||||
{{{ B01 }}} | {{{ B02 }}} | ||||||||||||||||||||||||||||||||||||||||||||||||
{{{ C01 }}} | {{{ C02 }}} | ||||||||||||||||||||||||||||||||||||||||||||||||
{{{ D01 }}} | {{{ D02 }}} | {{{ D03 }}} | |||||||||||||||||||||||||||||||||||||||||||||||
{{{ F01 }}} | {{{ F02 }}} | {{{ F03}}} | |||||||||||||||||||||||||||||||||||||||||||||||
{{{ G01 }}} | {{{ G02 }}} | {{{ G03 }}} | {{{ G04 }}} | ||||||||||||||||||||||||||||||||||||||||||||||
Treatment
Shown below is an algorithm summarizing the treatment of Abdominal aortic aneurysm according the the [...] guidelines.
Do's
Don'ts
- ↑ Singh, K.; Bønaa, KH.; Jacobsen, BK.; Bjørk, L.; Solberg, S. (2001). "Prevalence of and risk factors for abdominal aortic aneurysms in a population-based study : The Tromsø Study". Am J Epidemiol. 154 (3): 236–44. PMID 11479188. Unknown parameter
|month=
ignored (help) - ↑ Santosa, F.; Schrader, S.; Nowak, T.; Luther, B.; Kröger, K.; Bufe, A. (2013). "Thoracal, abdominal and thoracoabdominal aortic aneurysm". Int Angiol. 32 (5): 501–5. PMID 23903309. Unknown parameter
|month=
ignored (help) - ↑ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set. St. Louis: Mosby. ISBN 1-4160-2999-0.
- ↑ Clifton MA (Nov 1977). "Familial abdominal aortic aneurysms". Br J Surg. 64 (11): 765–6. doi:10.1002/bjs.1800641102. PMID 588966.
- ↑ Josselin-Mahr, L.; El Hessen, TA.; Toledano, C.; Fardet, L.; Kettaneh, A.; Tiev, K.; Cabane, J. (2013). "[Inflammatory aortitis in giant cell arteritis]". Presse Med. 42 (2): 151–9. doi:10.1016/j.lpm.2012.03.003. PMID 22552044. Unknown parameter
|month=
ignored (help) - ↑ Greenhalgh RM, Powell JT. "Endovascular repair of abdominal aortic aneurysm". N. Engl. J. Med. 358 (5): 494–501. doi:10.1056/NEJMct0707524. PMID 18234753.