Sandbox/table: Difference between revisions

Jump to navigation Jump to search
Guillermo Rodriguez Nava (talk | contribs)
No edit summary
Guillermo Rodriguez Nava (talk | contribs)
No edit summary
Line 12: Line 12:
|}
|}


==table==


{| style="border: 2px solid #DCDCDC; font-size: 90%; width: 80%;"
{| style="border: 2px solid #DCDCDC; font-size: 90%; width: 50%;"
|+ '''Laboratory findings'''
|+ '''Laboratory findings'''
|-
|-

Revision as of 18:59, 20 June 2014

The following table contains the main risk factors for CDI:[1][2][3][2][2]

Alterations in the coagulation system
Consumption of clotting factors
Increased concentrations of fibrin degradation products
Disseminated intravascular coagulation

table

Laboratory findings
Test Findings
White blood cells count Leucopenia (1000 cells/μL), with lymphopenia and neutrophilia
Blood smear Left shift with atypical lymphocytes
Liver function tests Raised aspartate aminotransferase and alanine aminotransferase, extended prothrombin time and partial thromboplastin time.
Proteins Hyperproteinemia.
Urinalysis Proteinuria.



table

Countries with a reported prevalence <15% of H. pylori resistance to clarithromycin
Diagnostic test North America South America Middle East Far East
ELISA (serology) detects:
  • Viral Antigen
  • IgM and IgG antibody | hol

There is a reported prevalence of 15% in the Northeast of the US.

  1. Hensgens MP, Goorhuis A, Dekkers OM, Kuijper EJ (2012). "Time interval of increased risk for Clostridium difficile infection after exposure to antibiotics". J Antimicrob Chemother. 67 (3): 742–8. doi:10.1093/jac/dkr508. PMID 22146873.
  2. 2.0 2.1 2.2 Knight, Christopher L.; Surawicz, Christina M. (2013). "Clostridium difficile Infection". Medical Clinics of North America. 97 (4): 523–536. doi:10.1016/j.mcna.2013.02.003. ISSN 0025-7125.
  3. Planche, Tim (2013). "Clostridium difficile". Medicine. 41 (11): 654–657. doi:10.1016/j.mpmed.2013.08.003. ISSN 1357-3039.