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==Postinflammatory hyperpigmentation== | |||
Also known as "Postinflammatory hypermelanosis can result from any natural or iatrogenic inflammatory condition, resulting from two mechanism: (1) increased epidermal pigmentation via increased [[melanocyte]] activity or (2) [[dermal]] [[melanosis]] from melanocyte damage and [[melanin]] drop out from the [[Epidermis (skin)|epidermis]] into the dermis.<ref name="Andrews">James, William; Berger, Timothy; Elston, Dirk (2005). ''Andrews' Diseases of the Skin: Clinical Dermatology''. (10th ed.). Saunders. ISBN 0-7216-2921-0.</ref>{{rp|854}}<ref name="Bolognia">{{cite book |author=Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. |title=Dermatology: 2-Volume Set |publisher=Mosby |location=St. Louis |year=2007 |pages= |isbn=1-4160-2999-0 |oclc= |doi= |accessdate=}}</ref> | |||
== See also == | == See also == | ||
* [[Postinflammatory hypopigmentation]] | * [[Postinflammatory hypopigmentation]] | ||
==References== | ==References== | ||
{{reflist}} | {{reflist|2}} | ||
Revision as of 17:52, 4 July 2014
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Postinflammatory hyperpigmentation
Also known as "Postinflammatory hypermelanosis can result from any natural or iatrogenic inflammatory condition, resulting from two mechanism: (1) increased epidermal pigmentation via increased melanocyte activity or (2) dermal melanosis from melanocyte damage and melanin drop out from the epidermis into the dermis.[1]:854[2]
See also
References
- ↑ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology. (10th ed.). Saunders. ISBN 0-7216-2921-0.
- ↑ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set. St. Louis: Mosby. ISBN 1-4160-2999-0.