Erythrasma pathophysiology

Erythrasma Microchapters
Home
Patient Information
Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Erythrasma from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
X Ray
CT
MRI
Echocardiography or Ultrasound
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Case #1
Erythrasma pathophysiology On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
slides
Images
American Roentgen Ray Society Images of Erythrasma pathophysiology All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Erythrasma pathophysiology
on Erythrasma pathophysiology
Erythrasma pathophysiology in the news
Blogs on Erythrasma pathophysiology
Directions to Hospitals Treating Erythrasma
Risk calculators and risk factors for Erythrasma pathophysiology

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Luke Rusowicz-Orazem, B.S.

Pathophysiology

Pathogenesis

• Erythrasma develops when Corynebacterium minitissium infiltrates the stratum corneum and proliferate.
  • The bacteria proliferates on the epidermis and develops at the intracellular level of the stratum corneum.
  • Penetration of the skin occurs when the horny cells of the plasma membranes rupture .
  • Keratolysis occurs due to the removal of keratin fibrils from the bacteria, leading to the formation of reddish-brown lesions characteristic of Erythrasma.