Metabolic alkalosis classification
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Marufa Marium, M.B.B.S[2]
Overview
Metabolic Alkalosis can be classified according to pathophysiology, etiology and chloride responsiveness.
Classification
The following classification of Metabolic Alkalosis is based on Pathophysiology:
{{Family tree | | | | A01 | | | |A01= Metabolic Alkalosis}| Stimulation on Collecting Duct | |||||||||||||||||||
| Primary stimulation by Mineralocorticoid Excess causing HTN: •Congenital Adrenal Hyperplasia •Cushing Syndrome •Primary aldosteronism •Renin Secreting tumors •Medications(Fluoroprednisolone, Fludrocortisone) •Liddle Syndrome •11 beta hydroxysteroid dehydrogenase deficiency | Secondary Stimulation by Chloride depletion causing normal or low blood pressure: •Vomiting •NG tube intubation •Congenital Chloridorrhea •Ileostomy •Cystic fibrosis •Diuretics •Bartter syndrome •Gitelman syndrome •Hypokalemia | ||||||||||||||||||