Total anomalous pulmonary venous connection risk factors
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Cafer Zorkun, M.D., Ph.D. [2]; Keri Shafer, M.D. [3]; Priyamvada Singh, MBBS[4]; Assistant Editor-In-Chief: Kristin Feeney, B.S. [5]
Overview
There are certain factors that increase the risk of congenital heart diseases.
Risk Factors
Risk factors during congenital heart diseases are:[1][2][3][4]
- Maternal obesity
- Medication during pregnancy
- Alcohol and drugs
- Rubella during pregnancy
- Diabetes during pregnancy
- Genetic factors
References
- ↑ Alvarado-Terrones, Evelyn G.; Perea-Cabrera, Maryangel; Klünder-Klünder, Miguel; Segura-Stanford, Begoña; Erdmenger-Orellana, Julio R.; Lopez-Yañez Blanco, Arturo; Hernández-Carbajal, Elizabeth; Granados Riverón, Javier T.; Mejía-Marín, Leonardo J.; Balderrabano-Saucedo, Norma A.; Contreras-Ramos, Alejandra; Díaz-Rosas, Guadalupe; Sánchez-Urbina, Rocío (2018). "Maternal Obesity as a Risk Factor for the Development of Total Anomalous Pulmonary Venous Connection in Their Offspring". Archives of Medical Research. 49 (2): 109–113. doi:10.1016/j.arcmed.2018.06.001. ISSN 0188-4409.
- ↑ Correa-Villaseñor, Adolfo; Ferencz, Charlotte; Boughman, Joann A.; Neill, Catherine A. (1991). "Total anomalous pulmonary venous return: Familial and environmental factors". Teratology. 44 (4): 415–428. doi:10.1002/tera.1420440408. ISSN 0040-3709.
- ↑ Phelan, K.; McDermid, H.E. (2011). "The 22q13.3 Deletion Syndrome (Phelan-McDermid Syndrome)". Molecular Syndromology. doi:10.1159/000334260. ISSN 1661-8777.
- ↑ Bleyl, Steven B.; Saijoh, Yukio; Bax, Noortje A.M.; Gittenberger-de Groot, Adriana C.; Wisse, Lambertus J.; Chapman, Susan C.; Hunter, Jennifer; Shiratori, Hidetaka; Hamada, Hiroshi; Yamada, Shigehito; Shiota, Kohei; Klewer, Scott E.; Leppert, Mark F.; Schoenwolf, Gary C. (2010). "Dysregulation of the PDGFRA gene causes inflow tract anomalies including TAPVR: integrating evidence from human genetics and model organisms". Human Molecular Genetics. 19 (7): 1286–1301. doi:10.1093/hmg/ddq005. ISSN 0964-6906.