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COVID-19 infection
 
 
 
 
 
 
 
 
 
 
 
ACE-2 receptor involvement
 
 
 
 
 
 
 
 
Increased Angiotensin II
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Altered coagulation biomarkers
 
 
 
 
 
COVID-19 Coagulopathy
 
 
 
 
 
Increased inflammatory markers
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Endothelial injury
 
 
 
 
 
 
 
 
Coagulation cacade activation
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Transient increase in antiphospholipid Abs