Bradycardia pathophysiology
|
Bradycardia Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Bradycardia pathophysiology On the Web |
|
American Roentgen Ray Society Images of Bradycardia pathophysiology |
|
Risk calculators and risk factors for Bradycardia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: M.Umer Tariq [2]
Overview
Pathophysiology
It is difficult and sometimes impossible to assign a mechanism to any particular bradycardia. However, the underlying mechanism is not clinically relevant to treatment, which is the same in both cases of sick sinus syndrome: a permanent pacemaker.
There are generally two types of problems that result in bradycardias:
Disorders of the sinus node
- Impaired automaticity - Sinus node dysfunction/sick sinus syndrome)[1]
- Exit block - Impaired conduction of the impulse from the sinus node into the surrounding atrial tissue
Disorders of the atrioventricular node (AV node)
- Atrioventricular conduction disturbances (1o AV block, 2o type I AV block, 2o type II AV block, 3o AV block) may result from impaired conduction in the AV node, or anywhere below it, such as in the bundle of HIS.
References
- ↑ Alpert MA, Flaker GC (1983). "Arrhythmias associated with sinus node dysfunction. Pathogenesis, recognition, and management". JAMA. 250 (16): 2160–6. PMID 6620520.