Psittacosis pathophysiology
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aditya Govindavarjhulla, M.B.B.S. [2]
Overview
Pathophysiology
Transmission The major risk factor for acquiring psittacosis is exposure to birds. Transmission can occur either by inhalation of aerosolized organisms in form of dried feces or respiratory secretions or by direct contact with birds[1]. As a result of this, the greatest incidence is seem in individuals with leisure or occupational exposure to birds. Cases of psittacosis can range from a sporadic case in a pet bird owner to an outbreak affecting several hundred birds in a commercial flock and multiple infected workers. A particular study showed that over 90% of fecal samples from birds (canaries, parrots, finches, budgerigars, pigeons, doves) caged in four aviaries in Turkey were polymerase chain reaction (PCR)-positive for C psittaci DNA. In an example of commercial C psittaci transmission, one study followed 20 turkeys and three people in real time, monitoring with serology, culture and PCR for 15 weeks. The turkeys
References
- ↑ Smith KA, Bradley KK, Stobierski MG, Tengelsen LA, National Association of State Public Health Veterinarians Psittacosis Compendium Committee (2005). "Compendium of measures to control Chlamydophila psittaci (formerly Chlamydia psittaci) infection among humans (psittacosis) and pet birds, 2005". J Am Vet Med Assoc. 226 (4): 532–9. PMID 15742693.