Renal tubular acidosis electrocardiogram
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Aditya Ganti M.B.B.S. [2]
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Electrocardiogram
Electrocardiogram changes in renal tubular acidosis is associated with potassium changes.
Hyperkalemia
EKG findings of hyperkalemia associated with renal tubular acidosis include:
- Peaked T Waves
- Peaked T waves are the earliest sign of hyperkalemia.
- Interventricular Conduction Delay
- It is observed when K > 6.5 meq/L
- Interventricular conduction delay is reflected by QRS widening that are inconsistent with LBBB or RBBB.
- Ventricular fibrillation
Hypokalemia
EKG findings of hypokalemia associated with renal tubular acidosis include:
- ST segment depression, decreased T wave amplitude, and prominent U waves:
- Prolongation of the QRS duration
- Increase in the amplitude and duration of the P-wave
- Cardiac arrhythmias and AV block
- Contrary to popular belief there is not prolongation of the QTc, this is artifactually prolonged due to the U wave. In some cases there is fusion of the T and U waves making interpretation impossible.
- Ventricular ectopy
References
- ↑ 1.0 1.1 Parham WA, Mehdirad AA, Biermann KM, Fredman CS (2006). "Hyperkalemia revisited". Tex Heart Inst J. 33 (1): 40–7. PMC 1413606. PMID 16572868.
- ↑ 2.0 2.1 2.2 Petrov DB (2012). "Images in clinical medicine. An electrocardiographic sine wave in hyperkalemia". N Engl J Med. 366 (19): 1824. doi:10.1056/NEJMicm1113009. PMID 22571204.
- ↑ Bonvini RF, Hendiri T, Anwar A (2006). "Sinus arrest and moderate hyperkalemia". Annales De Cardiologie Et D'angéiologie. 55 (3): 161–3. PMID 16792034. Unknown parameter
|month=ignored (help) - ↑ Mehta NJ, Chhabra VK, Khan IA (2001). "Sinus arrest or sinoventricular conduction in mild hyperkalemia". J Emerg Med. 20 (2): 163–4. PMID 11207412.