Premature ventricular contraction medical therapy
|
Premature ventricular contraction Microchapters |
|
Differentiating Premature Ventricular Contraction from other Disorders |
|---|
|
Diagnosis |
|
Treatment |
|
Case Studies |
|
Premature ventricular contraction medical therapy On the Web |
|
Premature ventricular contraction medical therapy in the news |
|
to Hospitals Treating Premature ventricular contraction medical therapy |
|
Risk calculators and risk factors for Premature ventricular contraction medical therapy |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Homa Najafi, M.D.[2] Mugilan Poongkunran M.B.B.S [3]
Overview
Isolated premature ventricular contractions with benign characteristics require no treatment. In healthy individuals, PVCs can often be resolved by restoring the balance of magnesium, calcium and potassium within the body.
Medical Therapy
- Pharmacological agents
- Antiarrhythmics: these agents alter the electrophysiologic mechanisms responsible for PVCs.
- Beta blocker : Propranolol, atenolol, metoprolol
- Calcium channel blockers
- Electrolytes replacement
- Magnesium supplements (e.g. magnesium citrate, orotate, Maalox, etc.)
- Potassium supplements
Therapies with limited data to support their use:
In the setting of existing cardiac disease, however, PVCs must be watched carefully, as they may cause a form of ventricular tachycardia (rapid heartbeat).
2017 AHA/ACC/HRS Guideline for Management of Patients with Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death (DO NOT EDIT)[1]
Recommendations for 12-lead ECG and Exercise Testing
| Class I |
| 1. In patients with VA symptoms associated with exertion, suspected ischemic heart disease, or catecholaminergic polymorphic ventricular tachycardia, exercise treadmill testing is useful to assess for exercise-induced VA (Level of Evidence: B-NR).[2][3]
2. In patients with suspected or documented VA, a 12-lead ECG should be obtained in sinus rhythm to look for evidence of heart disease (Level of Evidence: B-NR).[4] |
References
- ↑ Al-Khatib, Sana M.; Stevenson, William G.; Ackerman, Michael J.; Bryant, William J.; Callans, David J.; Curtis, Anne B.; Deal, Barbara J.; Dickfeld, Timm; Field, Michael E.; Fonarow, Gregg C.; Gillis, Anne M.; Granger, Christopher B.; Hammill, Stephen C.; Hlatky, Mark A.; Joglar, José A.; Kay, G. Neal; Matlock, Daniel D.; Myerburg, Robert J.; Page, Richard L. (2018). "2017 AHA/ACC/HRS Guideline for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death". Circulation. 138 (13). doi:10.1161/CIR.0000000000000549. ISSN 0009-7322.
- ↑ Elhendy, Abdou; Chandrasekaran, Krishnaswamy; Gersh, Bernard J; Mahoney, Douglas; Burger, Kelli N; Pellikka, Patricia A (2002). "Functional and prognostic significance of exercise-induced ventricular arrhythmias in patients with suspected coronary artery disease". The American Journal of Cardiology. 90 (2): 95–100. doi:10.1016/S0002-9149(02)02428-1. ISSN 0002-9149.
- ↑ Grady, Thomas A. (1998). "Prognostic Significance of Exercise-Induced Left Bundle-Branch Block". JAMA. 279 (2): 153. doi:10.1001/jama.279.2.153. ISSN 0098-7484.
- ↑ Pérez-Rodon, Jordi; Martínez-Alday, Jesus; Barón-Esquivias, Gonzalo; Martín, Alfonso; García-Civera, Roberto; del Arco, Carmen; Cano-Gonzalez, Alicia; Moya-Mitjans, Àngel (2014). "Prognostic value of the electrocardiogram in patients with syncope: Data from the Group for Syncope Study in the Emergency Room (GESINUR)". Heart Rhythm. 11 (11): 2035–2044. doi:10.1016/j.hrthm.2014.06.037. ISSN 1547-5271.