Tricuspid atresia natural history, complications and prognosis

	Tricuspid atresia Microchapters
Home
Patient Information
Overview
Pathophysiology
Causes
Differentiating Tricuspid Atresia from other Disorders
Epidemiology and Demographics
Risk Factors
Natural History, Complications and Prognosis
Diagnosis
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
Chest X Ray
CT
MRI
Echocardiography
Cardiac Catheterization
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Special Scenarios
Pregnancy
Case Studies
Case #1
Tricuspid atresia natural history, complications and prognosis On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
Powerpoint slides
Images
American Roentgen Ray Society Images of Tricuspid atresia natural history, complications and prognosis All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Tricuspid atresia natural history, complications and prognosis
CDC on Tricuspid atresia natural history, complications and prognosis
Tricuspid atresia natural history, complications and prognosis in the news
Blogs on Tricuspid atresia natural history, complications and prognosis
Directions to Hospitals Treating Type page name here
Risk calculators and risk factors for Tricuspid atresia natural history, complications and prognosis

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief:Sara Zand, M.D.[2] Keri Shafer, M.D. [3] Priyamvada Singh, MBBS [4] Assistant Editor-In-Chief: Kristin Feeney, B.S. [5]

Natural History, Complications and Prognosis

Early clinical features in infants include cyanosis of lips and tongue, difficulty in breathing, tiring easily during feeding.
The severity of cyanosis ininfants with pulmonary stenosis is dependent on the amount of pulmonary blood flow passing throughpatent ductus arteriosus.
After physiologic closure of patent ductus arteriosus (PDA), the cyanosis will be aggravated.
In patients with normal pulmonary blood flow, complications of heart failure may occur.
Prognosis is generally poor without surgery and 90% of patients will die before 10 years old.
The 15-year survival of patients with Fontan procedure is approximately %92 according to Merry et al.
- Complications of Fontan procedure include: